1. Tranilast Blunts the Hypertrophic and Fibrotic Response to Increased Afterload Independent of Cardiomyocyte Transient Receptor Potential Vanilloid 2 Channels
    Sheryl E. Koch et al, 2018, Journal of Cardiovascular Pharmacology CrossRef
  2. Activated fibroblasts in cardiac and cancer fibrosis: An overview of analogies and new potential therapeutic options
    Giuseppina Nicolini et al, 2023, Life Sciences CrossRef

  3. Tranilast Inhibits Pulmonary Fibrosis by Suppressing TGFβ/SMAD2 Pathway


    Motoyasu Kato et al, 2020, Drug Design, Development and Therapy CrossRef