1. Tilianin Protects against Ischemia/Reperfusion-Induced Myocardial Injury through the Inhibition of the Ca2+/Calmodulin-Dependent Protein Kinase II-Dependent Apoptotic and Inflammatory Signaling Pathways
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2. CaMKII, ‘jack of all trades’ in inflammation during cardiac ischemia/reperfusion injury
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3. Exercise-induced signaling pathways to counteracting cardiac apoptotic processes
   Hamed Alizadeh Pahlavani, 2022, Frontiers in Cell and Developmental Biology CrossRef
4. CaMKII‐dependent ryanodine receptor phosphorylation mediates sepsis‐induced cardiomyocyte apoptosis
   Marisa Sepúlveda et al, 2020, Journal of Cellular and Molecular Medicine CrossRef
5. Non-canonical WNT signalling in cardiovascular disease: mechanisms and therapeutic implications
   Ioannis Akoumianakis et al, 2022, Nature Reviews Cardiology CrossRef
6. Long-term administration of recombinant canstatin prevents adverse cardiac remodeling after myocardial infarction
   Akira Sugiyama et al, 2020, Scientific Reports CrossRef
7. Non-coding RNAs are involved in the response to oxidative stress
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8. CRABP1 in Non-Canonical Activities of Retinoic Acid in Health and Diseases
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9. Impact and potential mechanism of effects of chronic moderate alcohol consumption on cardiac function in aldehyde dehydrogenase 2 gene heterozygous mice
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10. The Importance of SGLT-2 Inhibitors as Both the Prevention and the Treatment of Diabetic Cardiomyopathy
    Klaudia Kowalska et al, 2022, Antioxidants CrossRef
11. Effects of exercise training and TrkB blockade on cardiac function and BDNF-TrkB signaling postmyocardial infarction in rats
    Heow Won Lee et al, 2018, American Journal of Physiology-Heart and Circulatory Physiology CrossRef
12. Ca2+/Calmodulin-Dependent Protein Kinase II Regulation by Inhibitor 1 of Protein Phosphatase 1 Protects Against Myocardial Ischemia–Reperfusion Injury
    Jin Yu et al, 2019, Journal of Cardiovascular Pharmacology and Therapeutics CrossRef
13. Levosimendan prevents doxorubicin-induced cardiotoxicity in time- and dose-dependent manner: implications for inotropy
    Panagiotis Efentakis et al, 2020, Cardiovascular Research CrossRef
14. The histone deacetylase inhibitor SAHA exerts a protective effect against myocardial ischemia/reperfusion injury by inhibiting sodium-calcium exchanger
    Xiaocui Shi et al, 2023, Biochemical and Biophysical Research Communications CrossRef
15. Increased oxidative stress and CaMKII activity contribute to electro‐mechanical defects in cardiomyocytes from a murine model of Huntington's disease
    Julliane Vasconcelos Joviano‐Santos et al, 2019, The FEBS Journal CrossRef
16. Gene Therapy for Catecholaminergic Polymorphic Ventricular Tachycardia by Inhibition of Ca 2+ /Calmodulin-Dependent Kinase II
    Vassilios J. Bezzerides et al, 2019, Circulation CrossRef
17. The hidden role of the Sigma1 receptor in muscle cells
    Michał Skrzycki et al, 2020, Journal of Receptors and Signal Transduction CrossRef
18. Combined fructose and sucrose consumption from an early age aggravates cardiac oxidative damage and causes a dilated cardiomyopathy in ‍SHR rats
    David Julian Arias-Chávez et al, 2023, Journal of Clinical Biochemistry and Nutrition CrossRef
19. Gene therapy for inherited arrhythmias
    Vassilios J Bezzerides et al, 2020, Cardiovascular Research CrossRef
20. Molecular basis for cellular retinoic acid-binding protein 1 in modulating CaMKII activation
    Jennifer Nhieu et al, 2023, Frontiers in Molecular Biosciences CrossRef
21. Proarrhythmic Remodeling of Calcium Homeostasis in Cardiac Disease; Implications for Diabetes and Obesity
    Shanna Hamilton et al, 2018, Frontiers in Physiology CrossRef
22. Calcium/Calmodulin-Dependent Protein Kinase II Delta Inhibition and Ventricular Remodeling After Myocardial Infarction
    Andrew J. Boyle et al, 2021, JAMA Cardiology CrossRef
23. Ca2+/calmodulin-dependent protein kinase II regulation by inhibitor 1 of protein phosphatase 1 alleviates necroptosis in high glucose-induced cardiomyocytes injury
    Linlin Sun et al, 2019, Biochemical Pharmacology CrossRef
24. Arsenic is more potent than cadmium or manganese in disrupting the INS-1 beta cell microRNA landscape
    Rowan Beck et al, 2019, Archives of Toxicology CrossRef
25. Reversing Cardiac Hypertrophy at the Source Using a Cardiac Targeting Peptide Linked to miRNA106a: Targeting Genes That Cause Cardiac Hypertrophy
    G. Ian Gallicano et al, 2022, Pharmaceuticals CrossRef