1. Interactions between iron and α-synuclein pathology in Parkinson's disease
    Bingbing Chen et al, 2019, Free Radical Biology and Medicine CrossRef
  2. PINK1 suppresses alpha-synuclein-induced neuronal injury: a novel mechanism in protein phosphatase 2A activation
    Weiwei Yang et al, 2018, Oncotarget CrossRef
  3. The Role of Alpha-Synuclein in Neurodevelopmental Diseases
    Sanaz Bordbar et al, 2024, Molecular Neurobiology CrossRef
  4. Alzheimer’s Disease and Dementia
    Michael Harper et al, 2020, Urological Care for Patients with Progressive Neurological Conditions CrossRef
  5. Loss of PINK1 inhibits apoptosis by upregulating α-synuclein in inflammation-sensitized hypoxic-ischemic injury in the immature brains
    Jianghu Zhu et al, 2016, Brain Research CrossRef
  6. Cytoprotection against Hypoxic and/or MPP+ Injury: Effect of δ–Opioid Receptor Activation on Caspase 3
    Yuan Xu et al, 2016, International Journal of Molecular Sciences CrossRef
  7. The delta‐opioid receptor and Parkinson’s disease
    Jin‐Zhong Huang et al, 2018, CNS Neuroscience & Therapeutics CrossRef
  8. Mitochondrial Inherited Disorders and their Correlation with Neurodegenerative Diseases
    Sofjana Gushi et al, 2024, Endocrine, Metabolic & Immune Disorders - Drug Targets CrossRef
  9. Dysregulation of Human Somatic piRNA Expression in Parkinson’s Disease Subtypes and Stages
    Tianjiao Zhang et al, 2022, International Journal of Molecular Sciences CrossRef
  10. La transconformation protéique, nouveau paradigme en neurologie
    J.-J. Hauw et al, 2015, Revue Neurologique CrossRef
  11. Liquid-liquid phase separation regulates alpha-synuclein aggregate and mitophagy in Parkinson’s disease
    Kaiying Hou et al, 2023, Frontiers in Neuroscience CrossRef
  12. The Lewy body dementias
    Richard R. Murphy et al, 2016, The Behavioral Neurology of Dementia CrossRef
  13. Alpha synuclein protein is involved in Aluminum-induced cell death and oxidative stress in PC12 cells
    Jamileh Saberzadeh et al, 2016, Brain Research CrossRef