1. CD69 partially inhibits apoptosis and erythroid differentiation via CD24, and their knockdown increase imatinib sensitivity in BCR‐ABL‐positive cells
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  2. Impact of Methimazole Treatment on Magnesium Concentration and Lymphocytes Activation in Adolescents with Graves’ Disease
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  3. NF-κB signaling pathway and its potential as a target for therapy in lymphoid neoplasms
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  4. B cell activation and proliferation increase intracellular zinc levels
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  5. Activin A downregulates the CD69-MT2A axis via p38MAPK to induce erythroid differentiation that sensitizes BCR-ABL-positive cells to imatinib
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