- Neuropilin-1 contributes to esophageal squamous cancer progression via promoting P65-dependent cell proliferation
F Shi et al, 2018, Oncogene CrossRef - Old Player-New Tricks: Non Angiogenic Effects of the VEGF/VEGFR Pathway in Cancer
Panagiotis Ntellas et al, 2020, Cancers CrossRef - Neuropilin-1: A Promising Therapeutic Target for Triple-Negative Breast Cancer
Noura Al-Zeheimi et al, 2024, Latest Research on Breast Cancer [Working Title] CrossRef - SPECT imaging of neuropilin receptor type-1 expression with 131I-labeled monoclonal antibody
Xiaofeng Dou et al, 2016, International Journal of Oncology CrossRef - Antiproliferative Modulation and Pro-Apoptotic Effect of BR2 Tumor-Penetrating Peptide Formulation 2-Aminoethyl Dihydrogen Phosphate in Triple-Negative Breast Cancer
Laertty Garcia de Sousa Cabral et al, 2023, Cancers CrossRef - Recent advances and future directions on small molecule VEGFR inhibitors in oncological conditions
Amandeep Thakur et al, 2024, European Journal of Medicinal Chemistry CrossRef - Neuropilins as Signaling Hubs, Controlling Tyrosine Kinases and Other Cell Surface Receptors
Sabrina Rizzolio et al, 2017, The Neuropilins: Role and Function in Health and Disease CrossRef - Vascular Endothelial Growth Factor-D (VEGF-D): An Angiogenesis Bypass in Malignant Tumors
Syeda Mahak Zahra Bokhari et al, 2023, International Journal of Molecular Sciences CrossRef - Viral gene therapy for breast cancer: progress and challenges
Antonela S. Asad et al, 2017, Expert Opinion on Biological Therapy CrossRef - Tumour growth and immune evasion as targets for a new strategy in advanced cancer
Andrea Nicolini et al, 2018, Endocrine-Related Cancer CrossRef - N-VEGF, the Autoregulatory Arm of VEGF-A
Marina Katsman et al, 2022, Cells CrossRef - NORAD-Regulated Signaling Pathways in Breast Cancer Progression
Ana Maria Capela et al, 2024, Cancers CrossRef - RNA binding protein Lin28B confers gastric cancer cells stemness via directly binding to NRP-1
Xiaocong Wang et al, 2018, Biomedicine & Pharmacotherapy CrossRef