1. Diallyl trisulfide suppresses doxorubicin‐induced cardiomyocyte apoptosis by inhibiting MAPK/NF‐κB signaling through attenuation of ROS generation
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  2. Aloe-emodin relieves zidovudine-induced injury in neonatal rat ventricular myocytes by regulating the p90rsk/p-bad/bcl-2 signaling pathway
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    Mi-Hua Liu et al, 2023, Molecular Medicine Reports CrossRef
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    Shuxu Du et al, 2018, Frontiers in Pharmacology CrossRef
  6. New NO- and H2S-releasing doxorubicins as targeted therapy against chemoresistance in castration-resistant prostate cancer: in vitro and in vivo evaluations
    Elisabetta Bigagli et al, 2018, Investigational New Drugs CrossRef
  7. Gp130-mediated STAT3 activation by S-propargyl-cysteine, an endogenous hydrogen sulfide initiator, prevents doxorubicin-induced cardiotoxicity
    J Wu et al, 2016, Cell Death & Disease CrossRef
  8. ERK: A Key Player in the Pathophysiology of Cardiac Hypertrophy
    Simona Gallo et al, 2019, International Journal of Molecular Sciences CrossRef
  9. H2S Donor SPRC Ameliorates Cardiac Aging by Suppression of JMJD3, a Histone Demethylase
    Sha Li et al, 2024, Antioxidants & Redox Signaling CrossRef
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  11. Exogenous hydrogen sulfide protects against high glucose‑induced apoptosis and oxidative stress by inhibiting the STAT3/HIF‑1α pathway in H9c2 cardiomyocytes
    Jing Li et al, 2019, Experimental and Therapeutic Medicine CrossRef
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