- Renoprotective Effects of Mineralocorticoid Receptor Antagonists Against Diabetic Kidney Disease
Sarah Bayne et al, 2024, Advanced Biology CrossRef - Integrin β3 overexpression contributes to podocyte injury through inhibiting RhoA/YAP signaling pathway
Zhuo Li et al, 2021, Bioengineered CrossRef - TGF-β1 modulates podocyte migration by regulating the expression of integrin-β1 and -β3 through different signaling pathways
Chien-An Chen et al, 2018, Biomedicine & Pharmacotherapy CrossRef - ISN Forefronts Symposium 2015: Nuclear Receptors and Diabetic Nephropathy
Bo Zheng et al, 2016, Kidney International Reports CrossRef - Mineralocorticoid Receptor Antagonists in Diabetic Kidney Disease
Daiji Kawanami et al, 2021, Frontiers in Pharmacology CrossRef - Role of the sodium‐hydrogen exchanger in mediating the renal effects of drugs commonly used in the treatment of type 2 diabetes
Milton Packer, 2018, Diabetes, Obesity and Metabolism CrossRef - A disease model of diabetic nephropathy in a glomerulus-on-a-chip microdevice
Li Wang et al, 2017, Lab on a Chip CrossRef - Role of Transient Receptor Potential Canonical Channel 6 (TRPC6) in Diabetic Kidney Disease by Regulating Podocyte Actin Cytoskeleton Rearrangement
Qian Wang et al, 2020, Journal of Diabetes Research CrossRef - Spironolactone alleviates diabetic nephropathy through promoting autophagy in podocytes
Dan Dong et al, 2019, International Urology and Nephrology CrossRef - HDAC6‐mediated α‐tubulin deacetylation suppresses autophagy and enhances motility of podocytes in diabetic nephropathy
Tiantian Liang et al, 2020, Journal of Cellular and Molecular Medicine CrossRef - Mineralocorticoid receptor antagonists in diabetic kidney disease — mechanistic and therapeutic effects
Jonatan Barrera-Chimal et al, 2022, Nature Reviews Nephrology CrossRef - Urolithin A Ameliorates the TGF Beta-Dependent Impairment of Podocytes Exposed to High Glucose
Barbara Lewko et al, 2024, Journal of Personalized Medicine CrossRef