1. MnTE-2-PyP, a manganese porphyrin, reduces cytotoxicity caused by irradiation in a diabetic environment through the induction of endogenous antioxidant defenses
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  2. Glycocalyx disruption enhances motility, proliferation and collagen synthesis in diabetic fibroblasts
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  3. Annexin A2 functions downstream of c-Jun N-terminal kinase to promote skin fibroblast cell migration
    Youpei Wang et al, 2017, Molecular Medicine Reports CrossRef
  4. Impact of mesenchymal stem cell-secretome-loaded hydrogel on proliferative and migratory activities of hyperglycemic fibroblasts
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  5. Nuclear factor-κB signaling negatively regulates high glucose-induced vascular endothelial cell damage downstream of the extracellular signal-regulated kinase/c-Jun N-terminal kinase pathway
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  6. Mitochondrial and metabolic remodelling in human skin fibroblasts in response to glucose availability
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  7. Role of fibroblast plasticity and heterogeneity in modulating angiogenesis and healing in the diabetic foot ulcer
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  8. Application of 3D Bioprinting Technologies to the Management and Treatment of Diabetic Foot Ulcers
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  10. Dermal Drivers of Injury-Induced Inflammation: Contribution of Adipocytes and Fibroblasts
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  11. Sonic hedgehog‑c‑Jun N‑terminal kinase‑zinc finger protein Gli1 signaling protects against high glucose concentration‑induced reactive oxygen species generation in human fibroblasts
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  12. Endothelial progenitor cell (EPCs)-derived exosomal miR-30d-5p inhibits the inflammatory response of high glucose-impaired fibroblasts by affecting the M1/M2 polarization of macrophages
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  13. Piper crocatum Ruiz & Pav. ameliorates wound healing through p53, E-cadherin and SOD1 pathways on wounded hyperglycemia fibroblasts
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  14. GSK3β-Ikaros-ANXA4 signaling inhibits high-glucose-induced fibroblast migration
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  15. The crosstalk of hedgehog, PI3K and Wnt pathways in diabetes
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  16. Effect of photobiomodulation at 830 nm on gene expression correlated with JAK/STAT signalling in wounded and diabetic wounded fibroblasts in vitro
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  18. Endogenous Biological Drivers in Diabetic Lower Limb Wounds Recurrence: Hypothetical Reflections
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