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2. Elucidating the Metabolic Plasticity of Cancer: Mitochondrial Reprogramming and Hybrid Metabolic States
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3. The enhancement of glycolysis regulates pancreatic cancer metastasis
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4. MiR-539 functions as a tumor suppressor in pancreatic cancer by targeting TWIST1
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6. Quantitative Proteomic Analysis in Alveolar Type II Cells Reveals the Different Capacities of RAS and TGF-β to Induce Epithelial–Mesenchymal Transition
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7. Endothelial to Mesenchymal Transition in Cardiovascular Disease
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8. Targeting mitochondria as a potential therapeutic strategy against chemoresistance in cancer
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9. Intrinsic and Extrinsic Modulators of the Epithelial to Mesenchymal Transition: Driving the Fate of Tumor Microenvironment
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10. Redox Imbalance in Idiopathic Pulmonary Fibrosis: A Role for Oxidant Cross-Talk Between NADPH Oxidase Enzymes and Mitochondria
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12. Metabolic reprogramming in breast cancer results in distinct mitochondrial bioenergetics between luminal and basal subtypes
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13. Functional Isolation of Tumor-Initiating Cells using Microfluidic-Based Migration Identifies Phosphatidylserine Decarboxylase as a Key Regulator
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14. Mitochondrial dysfunction induces the invasive phenotype, and cell migration and invasion, through the induction of AKT and AMPK pathways in lung cancer cells
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15. Biological and Clinical Impacts of Glucose Metabolism in Pancreatic Ductal Adenocarcinoma
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16. Deficiency of primary cilia in kidney epithelial cells induces epithelial to mesenchymal transition
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17. Krüppel-like factor 9 suppressed tumorigenicity of the pancreatic ductal adenocarcinoma by negatively regulating frizzled-5
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18. Correlation between S100A11 and the TGF-β1/SMAD4 pathway and its effects on the proliferation and apoptosis of pancreatic cancer cell line PANC-1
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