- Complement System: Promoter or Suppressor of Cancer Progression?
Margot Revel et al, 2020, Antibodies CrossRef - Protein Corona Gold Nanoparticles Fingerprinting Reveals a Profile of Blood Coagulation Proteins in the Serum of HER2-Overexpressing Breast Cancer Patients
María del Pilar Chantada-Vázquez et al, 2020, International Journal of Molecular Sciences CrossRef - A bacterial tyrosine phosphatase modulates cell proliferation through targeting RGCC
Chengcheng Liu et al, 2021, PLOS Pathogens CrossRef - Complement Inhibition: A Novel Form of Immunotherapy for Colon Cancer
Stephanie Downs-Canner et al, 2016, Annals of Surgical Oncology CrossRef - RGC32 induces epithelial-mesenchymal transition by activating the Smad/Sip1 signaling pathway in CRC
Xiao-Yan Wang et al, 2017, Scientific Reports CrossRef - Regulator of Cell Cycle Protein (RGCC/RGC-32) Protects against Pulmonary Fibrosis
Irina G. Luzina et al, 2022, American Journal of Respiratory Cell and Molecular Biology CrossRef - Complement and Cancer—A Dysfunctional Relationship?
Joshua M. Thurman et al, 2020, Antibodies CrossRef - Response gene to complement‑32 promotes cell survival via the NF‑κB pathway in non‑small‑cell lung cancer
Jing Zhang et al, 2019, Experimental and Therapeutic Medicine CrossRef - Regulation mechanism of PDK1 on macrophage metabolism and function
Yueqin Yang et al, 2016, Cell Biochemistry and Function CrossRef - Evasion and interactions of the humoral innate immune response in pathogen invasion, autoimmune disease, and cancer
Trisha A. Rettig et al, 2015, Clinical Immunology CrossRef - A review of human diseases caused or exacerbated by aberrant complement activation
Patrick L. McGeer et al, 2017, Neurobiology of Aging CrossRef - Contribution of the anaphylatoxin receptors, C3aR and C5aR, to the pathogenesis of pulmonary fibrosis
Hongmei Gu et al, 2016, The FASEB Journal CrossRef - RGC-32 induces transition of pancreatic cancer to epithelial mesenchyme in vivo
Liang Zhu et al, 2018, Pancreatology CrossRef - RGC-32 and diseases: the first 20 years
Sonia I. Vlaicu et al, 2019, Immunologic Research CrossRef - High Level of Anaphylatoxin C5a Predicts Poor Clinical Outcome in Patients with Clear Cell Renal Cell Carcinoma
Wei Xi et al, 2016, Scientific Reports CrossRef - Le système du complément
Marie Daugan et al, 2017, médecine/sciences CrossRef - Pumilio directs deadenylation-associated translational repression of the cyclin-dependent kinase 1 activator RGC-32
Michèle Brocard et al, 2018, Nucleic Acids Research CrossRef - Oncogenic gene RGC‐32 is a direct target of miR‐26b and facilitates tongue squamous cell carcinoma aggressiveness through EMT and PI3K/AKT signalling
Zhong‐Heng Yang et al, 2020, Cell Biochemistry and Function CrossRef - By Increasing the Expression and Activation of STAT3, Sustained C5a Stimulation Increases the Proliferation, Migration, and Invasion of RCC Cells and Promotes the Growth of Transgrafted Tumors
Jing-Min Zheng et al, 2021, Cancer Management and Research CrossRef - Complement C5a/C5aR pathway potentiates the pathogenesis of gastric cancer by down-regulating p21 expression
Jian Chen et al, 2018, Cancer Letters CrossRef - C5a induces A549 cell proliferation of non-small cell lung cancer via GDF15 gene activation mediated by GCN5-dependent KLF5 acetylation
Chenhui Zhao et al, 2018, Oncogene CrossRef - Enhancement of cancer invasion and growth via the C5a‑C5a receptor system: Implications for cancer promotion by autoimmune diseases and association with cervical cancer invasion
Masakazu Yoneda et al, 2018, Oncology Letters CrossRef - Expression of RGC32 in human normal and preeclamptic placentas and its role in trophoblast cell invasion and migration
Q.-j. Wang et al, 2015, Placenta CrossRef - Anaphylatoxins Enhance Recruitment of Nonclassical Monocytes via Chemokines Produced by Pleural Mesothelial Cells in Tuberculous Pleural Effusion
Lisha Luo et al, 2019, American Journal of Respiratory Cell and Molecular Biology CrossRef - Complementing the Cancer-Immunity Cycle
Ruben Pio et al, 2019, Frontiers in Immunology CrossRef