1
|
Deveraux QL, Takahashi R, Salvesen GS and
Reed JC: X-linked IAP is a direct inhibitor of cell-death
proteases. Nature. 388:300–304. 1997. View
Article : Google Scholar : PubMed/NCBI
|
2
|
Chen G: The relationship between the
expression of TAM, survivin and the degree of necrosis of the tumor
after cisplatin treatment in osteosarcoma. Eur Rev Med Pharmacol
Sci. 21:490–497. 2017.PubMed/NCBI
|
3
|
Musacchio A: Spindle assembly checkpoint:
The third decade. Philos Trans R Soc Lond B Biol Sci.
366:3595–3604. 2011. View Article : Google Scholar : PubMed/NCBI
|
4
|
Kuo ML, Shen SC, Yang CH, Chuang SE, Cheng
AL and Huang TS: Bcl-2 prevents topoisomerase II inhibitor
GL331-induced apoptosis is mediated by down-regulation of
poly(ADP-ribose)polymerase activity. Oncogene. 17:2225–2234. 1998.
View Article : Google Scholar : PubMed/NCBI
|
5
|
O'Connor DS, Grossman D, Plescia J, Li F,
Zhang H, Villa A, Tognin S, Marchisio PC and Altieri DC: Regulation
of apoptosis at cell division by p34cdc2 phosphorylation of
survivin. Proc Natl Acad Sci USA. 97:pp. 13103–13107. 2000;
View Article : Google Scholar : PubMed/NCBI
|
6
|
Shin S, Sung BJ, Cho YS, Kim HJ, Ha NC,
Hwang JI, Chung CW, Jung YK and Oh BH: An anti-apoptotic protein
human survivin is a direct inhibitor of caspase-3 and −7.
Biochemistry. 40:1117–1123. 2001. View Article : Google Scholar : PubMed/NCBI
|
7
|
Suzuki A, Ito T, Kawano H, Hayashida M,
Hayasaki Y, Tsutomi Y, Akahane K, Nakano T, Miura M and Shiraki K:
Survivin initiates procaspase 3/p21 complex formation as a result
of interaction with Cdk4 to resist Fas-mediated cell death.
Oncogene. 19:1346–1353. 2000. View Article : Google Scholar : PubMed/NCBI
|
8
|
Olie RA, Simões-Wüst AP, Baumann B, Leech
SH, Fabbro D, Stahel RA and Zangemeister-Wittke U: A novel
antisense oligonucleotide targeting survivin expression induces
apoptosis and sensitizes lung cancer cells to chemotherapy. Cancer
Res. 60:2805–2809. 2000.PubMed/NCBI
|
9
|
Tamm I, Wang Y, Sausville E, Scudiero DA,
Vigna N, Oltersdorf T and Reed JC: IAP-family protein survivin
inhibits caspase activity and apoptosis induced by Fas (CD95), Bax,
caspases, and anticancer drugs. Cancer Res. 58:5315–5320.
1998.PubMed/NCBI
|
10
|
Kappler M, Köhler T, Kampf C,
Diestelkötter P, Würl P, Schmitz M, Bartel F, Lautenschläger C,
Rieber EP, Schmidt H, et al: Increased survivin transcript levels:
An independent negative predictor of survival in soft tissue
sarcoma patients. Int J Cancer. 95:360–363. 2001.PubMed/NCBI
|
11
|
Pui CH and Evans WE: Treatment of acute
lymphoblastic leukemia. N Engl J Med. 354:166–178. 2006. View Article : Google Scholar : PubMed/NCBI
|
12
|
Campana D, Coustan-Smith E, Manabe A,
Buschle M, Raimondi SC, Behm FG, Ashmun R, Aricò M, Biondi A and
Pui CH: Prolonged survival of B-lineage acute lymphoblastic
leukemia cells is accompanied by overexpression of bcl-2 protein.
Blood. 81:1025–1031. 1993.PubMed/NCBI
|
13
|
Flotho C, Coustan-Smith E, Pei D, Iwamoto
S, Song G, Cheng C, Pui CH, Downing JR and Campana D: Genes
contributing to minimal residual disease in childhood acute
lymphoblastic leukemia: Prognostic significance of CASP8AP2. Blood.
108:1050–1057. 2006. View Article : Google Scholar : PubMed/NCBI
|
14
|
Jaskoll T, Chen H, Min Zhou Y, Wu D and
Melnick M: Developmental expression of survivin during embryonic
submandibular salivary gland development. BMC Dev Biol. 1:52001.
View Article : Google Scholar : PubMed/NCBI
|
15
|
Pui CH, Relling MV and Downing JR: Acute
lymphoblastic leukemia. N Engl J Med. 350:1535–1548. 2004.
View Article : Google Scholar : PubMed/NCBI
|
16
|
Krajinovic M, Labuda D, Richer C, Karimi S
and Sinnett D: Susceptibility to childhood acute lymphoblastic
leukemia: Influence of CYP1A1, CYP2D6, GSTM1, and GSTT1 genetic
polymorphisms. Blood. 93:1496–1501. 1999.PubMed/NCBI
|
17
|
Canalle R, Burim RV, Tone LG and Takahashi
CS: Genetic polymorphisms and susceptibility to childhood acute
lymphoblastic leukemia. Environ Mol Mutagen. 43:100–109. 2004.
View Article : Google Scholar : PubMed/NCBI
|
18
|
Lanciotti M, Dufour C, Corral L, Di
Michele P, Pigullo S, De Rossi G, Basso G, Leszl A, Luciani M, Lo
Nigro L, et al: Genetic polymorphism of NAD(P)H:quinone
oxidoreductase is associated with an increased risk of infant acute
lymphoblastic leukemia without MLL gene rearrangements. Leukemia.
19:214–216. 2005. View Article : Google Scholar : PubMed/NCBI
|
19
|
Bolufer P, Barragan E, Collado M, Cervera
J, López JA and Sanz MA: Influence of genetic polymorphisms on the
risk of developing leukemia and on disease progression. Leuk Res.
30:1471–1491. 2006. View Article : Google Scholar : PubMed/NCBI
|
20
|
Aydin-Sayitoglu M, Hatirnaz O, Erensoy N
and Ozbek U: Role of CYP2D6, CYP1A1, CYP2E1, GSTT1, and GSTM1 genes
in the susceptibility to acute leukemias. Am J Hematol. 81:162–170.
2006. View Article : Google Scholar : PubMed/NCBI
|
21
|
d'Errico A, Mamo C, Costa G, Filippi M and
Crosignani P: Use of pension records for occupational health
surveillance: Example of record-linkage with hospital discharge
records to study the association between work and the incidence of
leukaemias, lung and bladder cancer, and miscarriage. Med Lav.
96(Suppl): s147–s160. 2005.(In Italian). PubMed/NCBI
|
22
|
LaCasse EC, Baird S, Korneluk RG and
MacKenzie AE: The inhibitors of apoptosis (IAPs) and their emerging
role in cancer. Oncogene. 17:3247–3259. 1998. View Article : Google Scholar : PubMed/NCBI
|
23
|
Gilbert ME and Shafer TJ: In vitro
exposure to aluminum does not alter long-term potentiation or
glutamate release in rat hippocampal slices. Neurotoxicol Teratol.
18:175–180. 1996. View Article : Google Scholar : PubMed/NCBI
|
24
|
Takashima A, Noguchi K, Michel G, Mercken
M, Hoshi M, Ishiguro K and Imahori K: Exposure of rat hippocampal
neurons to amyloid beta peptide (25–35) induces the inactivation of
phosphatidyl inositol-3 kinase and the activation of tau protein
kinase I/glycogen synthase kinase-3 beta. Neurosci Lett. 203:33–36.
1996. View Article : Google Scholar : PubMed/NCBI
|
25
|
Mullikin JC, Hunt SE, Cole CG, Mortimore
BJ, Rice CM, Burton J, Matthews LH, Pavitt R, Plumb RW, Sims SK, et
al: An SNP map of human chromosome 22. Nature. 407:516–520. 2000.
View Article : Google Scholar : PubMed/NCBI
|
26
|
Altshuler D, Pollara VJ, Cowles CR, Van
Etten WJ, Baldwin J, Linton L and Lander ES: An SNP map of the
human genome generated by reduced representation shotgun
sequencing. Nature. 407:513–516. 2000. View
Article : Google Scholar : PubMed/NCBI
|
27
|
Gray IC, Campbell DA and Spurr NK: Single
nucleotide polymorphisms as tools in human genetics. Hum Mol Genet.
9:2403–2408. 2000. View Article : Google Scholar : PubMed/NCBI
|
28
|
Carlson CS, Eberle MA, Rieder MJ, Yi Q,
Kruglyak L and Nickerson DA: Selecting a maximally informative set
of single-nucleotide polymorphisms for association analyses using
linkage disequilibrium. Am J Hum Genet. 74:106–120. 2004.
View Article : Google Scholar : PubMed/NCBI
|
29
|
Nowotny P, Kwon JM and Goate AM: SNP
analysis to dissect human traits. Curr Opin Neurobiol. 11:637–641.
2001. View Article : Google Scholar : PubMed/NCBI
|
30
|
Jang JS, Kim KM, Kang KH, Choi JE, Lee WK,
Kim CH, Kang YM, Kam S, Kim IS, Jun JE, et al: Polymorphisms in the
survivin gene and the risk of lung cancer. Lung Cancer. 60:31–39.
2008. View Article : Google Scholar : PubMed/NCBI
|