Related Articles
Both HPV and carcinogen contribute to the development of resistance to apoptosis during oral carcinogenesis.
Conversion of normal human oral keratinocytes to tumorigenic cells is associated with the acquisition of resistance to TGF-beta.
Activity of human papillomavirus type 16 P97 promoter in immortal and tumorigenic human oral keratinocytes.
HIV-1 Tat enhances replicative potential of human oral keratinocytes harboring HPV-16 genome
Increased ICAM-1 expression in transformed human oral epithelial cells: molecular mechanism and functional role in peripheral blood mononuclear cell adhesion and lymphokine-activated-killer cell cytotoxicity.