Related Articles
Resveratrol inhibits doxorubicin‑induced cardiotoxicity via sirtuin 1 activation in H9c2 cardiomyocytes
Hydrogen sulfide attenuates doxorubicin-induced cardiotoxicity by inhibiting reactive oxygen species-activated extracellular signal-regulated kinase 1/2 in H9c2 cardiac myocytes
Attenuation of doxorubicin-induced cardiotoxicity by esculetin through modulation of Bmi-1 expression
Resveratrol prevents doxorubicin-induced cardiotoxicity in H9c2 cells through the inhibition of endoplasmic reticulum stress and the activation of the Sirt1 pathway
Mitochondria‑associated protein LRPPRC exerts cardioprotective effects against doxorubicin‑induced toxicity, potentially via inhibition of ROS accumulation