1
|
Ferlay J, Soerjomataram I, Ervik M,
Dikshit R, Eser S, Mathers C, Rebelo M, Parkin DM, Forman D and
Bray F: GLOBOCAN 2012 v10, Cancer Incidence and Mortality
Worldwide: IARC CancerBase No. 11 [Internet]. Lyon, France:
International Agency for Research on Cancer; 2013, http://globocan.iarc.fr.
Accessed December 18, 2013
|
2
|
Bidard FC, Mathiot C, Delaloge S, et al:
Single circulating tumor cell detection and overall survival in
nonmetastatic breast cancer. Ann Oncol. 21:729–733. 2010.
View Article : Google Scholar : PubMed/NCBI
|
3
|
Cristofanilli M, Budd GT, Ellis MJ, et al:
Circulating tumor cells, disease progression, and survival in
metastatic breast cancer. N Engl J Med. 351:781–791. 2004.
View Article : Google Scholar : PubMed/NCBI
|
4
|
Hayes DF, Cristofanilli M, Budd GT, et al:
Circulating tumor cells at each follow-up time point during therapy
of metastatic breast cancer patients predict progression-free and
overall survival. Clin Cancer Res. 12:4218–4224. 2006. View Article : Google Scholar
|
5
|
Pantel K and Alix-Panabières C:
Circulating tumour cells in cancer patients: challenges and
perspectives. Trends Mol Med. 16:398–406. 2010. View Article : Google Scholar : PubMed/NCBI
|
6
|
Chambers AF, Groom AC and MacDonald IC:
Dissemination and growth of cancer cells in metastatic sites. Nat
Rev Cancer. 2:563–572. 2002. View
Article : Google Scholar : PubMed/NCBI
|
7
|
Thiery JP: Epithelial-mesenchymal
transitions in tumour progression. Nat Rev Cancer. 2:442–454. 2002.
View Article : Google Scholar : PubMed/NCBI
|
8
|
Gheldof A and Berx G: Cadherins and
epithelial-to-mesenchymal transition. Prog Mol Biol Transl Sci.
116:317–336. 2013. View Article : Google Scholar : PubMed/NCBI
|
9
|
Martinez-Alvarez C, Blanco MJ, Perez R, et
al: Snail family members and cell survival in physiological and
pathological cleft palates. Dev Biol. 265:207–218. 2004. View Article : Google Scholar : PubMed/NCBI
|
10
|
Yu W, Kamara H and Svoboda KK: The role of
twist during palate development. Dev Dyn. 237:2716–2725. 2008.
View Article : Google Scholar : PubMed/NCBI
|
11
|
Yu M, Bardia A, Wittner BS, et al:
Circulating breast tumor cells exhibit dynamic changes in
epithelial and mesenchymal composition. Science. 339:580–584. 2013.
View Article : Google Scholar : PubMed/NCBI
|
12
|
Williams LV, Veliceasa D, Vinokour E and
Volpert OV: miR-200b inhibits prostate cancer EMT, growth and
metastasis. PLoS One. 8:e839912013. View Article : Google Scholar : PubMed/NCBI
|
13
|
Virtakoivu R, Pellinen T, Rantala JK,
Perälä M and Ivaska J: Distinct roles of AKT isoforms in regulating
β1-integrin activity, migration, andinvasion in prostate cancer.
Mol Biol Cell. 23:3357–3369. 2012.
|
14
|
Ren J, Chen Y, Song H, Chen L and Wang R:
Inhibition of ZEB1 reverses EMT and chemoresistance in
docetaxel-resistant human lung adenocarcinoma cell line. J Cell
Biochem. 114:1395–1403. 2013. View Article : Google Scholar : PubMed/NCBI
|
15
|
Rosano L, Cianfrocca R, Spinella F, et al:
Acquisition of chemoresistance and EMT phenotype is linked with
activation of the endothelin A receptor pathway in ovarian
carcinoma cells. Clin Cancer Res. 17:2350–2360. 2011. View Article : Google Scholar : PubMed/NCBI
|
16
|
Guarino M, Rubino B and Ballabio G: The
role of epithelial-mesenchymal transition in cancer pathology.
Pathology. 39:305–318. 2007. View Article : Google Scholar : PubMed/NCBI
|
17
|
Gao Q, Liu W, Cai J, Li M, Gao Y, Lin W
and Li Z: EphB2 promotes cervical cancer progression by inducing
epithelial-mesenchymal transition. Hum Pathol. 45:372–381. 2014.
View Article : Google Scholar : PubMed/NCBI
|
18
|
Mayer A, Hockel M, Schlischewsky N,
Schmidberger H, Horn LC and Vaupel P: Lacking hypoxia-mediated
downregulation of E-cadherin in cancers of the uterine cervix. Br J
Cancer. 108:402–408. 2013. View Article : Google Scholar : PubMed/NCBI
|
19
|
Pichler M, Ress AL, Winter E, et al:
MiR-200a regulates epithelial to mesenchymal transition-related
gene expression and determines prognosis in colorectal cancer
patients. Br J Cancer. 110:1614–1621. 2014. View Article : Google Scholar : PubMed/NCBI
|
20
|
Raimondi C, Gradilone A, Naso G, et al:
Epithelial-mesenchymal transition and stemness features in
circulating tumor cells from breast cancer patients. Breast Cancer
Res Treat. 130:449–455. 2011. View Article : Google Scholar : PubMed/NCBI
|
21
|
Armstrong AJ, Marengo MS, Oltean S, et al:
Circulating tumor cells from patients with advanced prostate and
breast cancer display both epithelial and mesenchymal markers. Mol
Cancer Res. 9:997–1007. 2011. View Article : Google Scholar : PubMed/NCBI
|
22
|
Ohashi S, Natsuizaka M, Naganuma S, et al:
A NOTCH3-mediated squamous cell differentiation program limits
expansion of EMT-competent cells that express the ZEB transcription
factors. Cancer Res. 71:6836–6847. 2011. View Article : Google Scholar : PubMed/NCBI
|
23
|
Maseki S, Ijichi K, Tanaka H, et al:
Acquisition of EMT phenotype in the gefitinib-resistant cells of a
head and neck squamous cell carcinoma cell line through
Akt/GSK-3β/snail signaling pathway. Br J Cancer. 106:1196–1204.
2012.PubMed/NCBI
|
24
|
Wei J, Li Z, Chen W, Ma C, Zhan F, Wu W
and Peng Y: AEG-1 participates in TGF-beta1-induced EMT through p38
MAPK activation. Cell Biol Int. 37:1016–1021. 2013. View Article : Google Scholar : PubMed/NCBI
|
25
|
Carpenter RL, Paw I, Dewhirst MW and Lo
HW: Akt phosphorylates and activates HSF-1 independent of heat
shock, leading to Slug overexpression and epithelial-mesenchymal
transition (EMT) of HER2-overexpressing breast cancer cells.
Oncogene. Jan 27–2014.(Epub ahead of print). View Article : Google Scholar
|
26
|
Okui G, Tobiume K, Rizqiawan A, et al: AKT
primes snail-induced EMT concomitantly with the collective
migration of squamous cell carcinoma cells. J Cell Biochem.
114:2039–2049. 2013. View Article : Google Scholar : PubMed/NCBI
|
27
|
Sanchez-Tillo E, de Barrios O, Siles L,
Cuatrecasas M, Castells A and Postigo A: beta-catenin/TCF4 complex
induces the epithelial-to-mesenchymal transition (EMT)-activator
ZEB1 to regulate tumor invasiveness. Proc Natl Acad Sci USA.
108:19204–19209. 2011. View Article : Google Scholar : PubMed/NCBI
|
28
|
Huang T, Chen Z and Fang L: Curcumin
inhibits LPS-induced EMT through downregulation of NF-kappaB-Snail
signaling in breast cancer cells. Oncol Rep. 29:117–124.
2013.PubMed/NCBI
|
29
|
Liu J, Ruan B, You N, et al:
Downregulation of miR-200a induces EMT phenotypes and CSC-like
signatures through targeting the β-catenin pathway in hepatic oval
cells. PLoS One. 8:e794092013.PubMed/NCBI
|
30
|
Cong N, Du P, Zhang A, et al:
Downregulated microRNA-200a promotes EMT and tumor growth through
the wnt/beta-catenin pathway by targeting the E-cadherin repressors
ZEB1/ZEB2 in gastric adenocarcinoma. Oncol Rep. 29:1579–1587.
2013.PubMed/NCBI
|
31
|
Zhao Y, Li X, Sun X, Zhang Y and Ren H:
EMT phenotype is induced by increased Src kinase activity via
Src-mediated caspase-8 phosphorylation. Cell Physiol Biochem.
29:341–352. 2012. View Article : Google Scholar : PubMed/NCBI
|
32
|
Wang H, Wang HS, Zhou BH, et al:
Epithelial-mesenchymal transit ion (EMT) induced by TNF-alpha
requires AKT/GSK-3beta-mediated stabilization of snail in
colorectal cancer. PLoS One. 8:e566642013. View Article : Google Scholar : PubMed/NCBI
|
33
|
Wu Y, Ginther C, Kim J, Mosher N, Chung S,
Slamon D and Vadgama JV: Expression of Wnt3 activates
Wnt/beta-catenin pathway and promotes EMT-like phenotype in
trastuzumab-resistant HER2-overexpressing breast cancer cells. Mol
Cancer Res. 10:1597–1606. 2012. View Article : Google Scholar : PubMed/NCBI
|
34
|
Mikaelian I, Malek M, Gadet R, et al:
Genetic and pharmacologic inhibition of mTORC1 promotes EMT by a
TGF-β-independent mechanism. Cancer Res. 73:6621–6631.
2013.PubMed/NCBI
|