[Corrigendum] miR‑382 inhibits breast cancer progression and metastasis by affecting the M2 polarization of tumor‑associated macrophages by targeting PGC‑1α

• Authors:
  • Hua Zhou
  • Mingyu Gan
  • Xin Jin
  • Meng Dai
  • Yuanyuan Wang
  • Youyang Lei
  • Zijing Lin
  • Jia Ming

• View Affiliations

  
  Affiliations: Department of Breast and Thyroid Surgery, The Affiliated Shapingba Hospital of Chongqing University, Chongqing 400030, P.R. China, Shanxi Medical University, Taiyuan, Shanxi 030607, P.R. China, Department of Critical Care Medicine, The Affiliated Fuling Hospital of Chongqing University, Chongqing 408099, P.R. China, Department of Breast and Thyroid Surgery, The Second Affiliated Hospital of Chongqing Medical University, Chongqing 400010, P.R. China
• Published online on: November 2, 2022     https://doi.org/10.3892/ijo.2022.5449
• Article Number: 1

• Copyright : © Zhou et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].

Article

Int J Oncol 61: [Related article:] 126, 2022; DOI : 10.3892/ijo.2022.5416

Subsequently to the publication of the above article, the authors have contacted the Editorial Office to explain that Fig. 7 was published containing an erroneously placed data panel. Specifically, the center panel of the images selected for the invasion assay experiments portrayed in Fig. 7A (i.e., the miR-382 experiment) was chosen incorrectly, and the authors have requested that this panel be replaced by the panel containing the data that was actually used for the statistical analysis shown in part (B).

Figure 7 PGC-1α reverses the changes in the biological characteristics of breast cancer cells induced by TAMs with high miR-382 expression. (A and B) Transwell invasion experiments were performed to investigate the invasion abilities of 4T1 cells in different groups (×200). (C and D) A wound healing assay was used to examine the migration of 4T1 cells (×100). (E and F) The expression levels of EMT markers (epithelial marker: E-cadherin; mesenchymal marker: vimentin) in 4T1 cells were detected using western blot analysis. GAPDH was used as the internal reference. Data are presented as the means ± SD of three independent experiments. *P<0.05 and **P<0.01.

The revised version of Fig. 7, showing the correct data panel for the miR-382 experiment in the invasion assay images in part (A), is shown on the next page. The authors can confirm that the change made to this figure does not affect the overall conclusions reported in the study, and all the authors agree to the publication of this corrigendum. The authors are grateful to the Editor of International Journal of Oncology for allowing them the opportunity to publish this additional Corrigendum; furthermore, they apologize for any inconvenience caused to the readership of the Journal.

Figure 7 PGC-1α reverses the changes in the biological characteristics of breast cancer cells induced by TAMs with high miR-382 expression. (A and B) Transwell invasion experiments were performed to investigate the invasion abilities of 4T1 cells in different groups (×200). (C and D) A wound healing assay was used to examine the migration of 4T1 cells (×100). (E and F) The expression levels of EMT markers (epithelial marker: E-cadherin; mesenchymal marker: vimentin) in 4T1 cells were detected using western blot analysis. GAPDH was used as the internal reference. Data are presented as the means ± SD of three independent experiments. *P<0.05 and **P<0.01.