[Corrigendum] The tetraspanin CD151‑ARSA mutant inhibits angiogenesis via the YRSL sequence

• Authors:
  • Dan Peng
  • Houjuan Zuo
  • Zhengxiang Liu
  • Jin Qin
  • Yuanlin Zhou
  • Pengcheng Li
  • Daowen Wang
  • Hesong Zeng
  • Xin A. Zhang

• View Affiliations

  
  Affiliations: Department of Nuclear Medicine, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, P.R. China, Department of Cardiology of Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, P.R. China, Department of Physiology and Cancer Center, University of Oklahoma Health Science Center, Oklahoma City, OK, USA
• Published online on: August 10, 2023     https://doi.org/10.3892/mmr.2023.13069
• Article Number: 182

• Copyright : © Peng et al. This is an open access article distributed under the terms of Creative Commons Attribution License [CC BY 4.0].

Article

Mol Med Rep 7: [Related article:] 836–842, 2013; DOI: 10.3892/mmr.2012.1250

Subsequently to the publication of the above paper, an interested reader drew to the authors’ attention that, in Fig. 4A on p. 839, the ‘CD151/24 h’ and ‘CD151-ARSA/48 h’ panels appeared to contain overlapping sections of data, such that they were potentially derived from the same original source, where these panels were intended to show the results from differently performed experiments. The authors have re-examined their original data, and realize that the ‘CD151-ARSA/48 h’ panel was inadvertently placed incorrectly in the figure.

Figure 4. Effects of CD151 and CD151-ARSA transfection on the capillary network formation of HUVECs. (A) Representative photomicrographs observed at 12, 24 and 48 h after rAAV transfection on Matrigel showed that HUVECs assembled into capillary network structures. (B) Quantitative analysis of capillary network formation. *p<0.05 vs. control and GFP group. #p<0.05 vs. CD151 group. HUVECs, human umbilical vein endothelial cells; GFP, green fluorescent protein.

The revised version of Fig. 4, now containing the correct data for the ‘CD151-ARSA/48 h’ experiment in Fig. 4A, is shown below. Note that this error did not adversely affect either the results or the overall conclusions reported in this study. All the authors agree with the publication of this corrigendum, and are grateful to the Editor of Molecular Medicine Reports for allowing them the opportunity to publish this. They also wish to apologize to the readership of the Journal for any inconvenience caused.