1
|
Shekar PS: Cardiology patient page.
On-pump and off-pump coronary artery bypass grafting. Circulation.
113:e51–e52. 2006. View Article : Google Scholar
|
2
|
Ehsan A, Shekar P and Aranki S: Innovative
surgical strategies: Minimally invasive CABG and off-pump CABG.
Curr Treat Options Cardiovasc Med. 6:43–51. 2004. View Article : Google Scholar : PubMed/NCBI
|
3
|
Palmerini T, Biondi-Zoccai G, Riva DD, et
al: Risk of stroke with percutaneous coronary intervention compared
with on-pump and off-pump coronary artery bypass graft surgery:
Evidence from a comprehensive network meta-analysis. Am Heart J.
165:910–917. 2013. View Article : Google Scholar
|
4
|
Harskamp RE, Lopes RD, Baisden CE, De
Winter RJ and Alexander JH: Saphenous vein graft failure after
coronary artery bypass surgery: pathophysiology, management and
future directions. Ann Surg. 257:824–833. 2013. View Article : Google Scholar : PubMed/NCBI
|
5
|
Bakaeen FG, Chu D, Kelly RF, et al:
Performing Coronary Artery Bypass Grafting Off-Pump May Compromise
Long-Term Survival in a Veteran Population. Ann Thorac Surg.
95:1952–1958. 2013. View Article : Google Scholar : PubMed/NCBI
|
6
|
Moller CH, Penninga L, Wetterslev J,
Steinbruchel DA and Gluud C: Off-pump versus on-pump coronary
artery bypass grafting for ischaemic heart disease. Cochrane
Database Syst Rev. 3:CD0072242012.PubMed/NCBI
|
7
|
Nathoe HM, Van Dijk D, Jansen EW, et al: A
comparison of on-pump and off-pump coronary bypass surgery in
low-risk patients. N Engl J Med. 348:394–402. 2003. View Article : Google Scholar : PubMed/NCBI
|
8
|
Orhan G, Sargin M, Senay S, et al:
Systemic and myocardial inflammation in traditional and off-pump
cardiac surgery. Tex Heart Inst J. 34:160–165. 2007.PubMed/NCBI
|
9
|
Selvanayagam JB, Petersen SE, Francis JM,
et al: Effects of off-pump versus on-pump coronary surgery on
reversible and irreversible myocardial injury: a randomized trial
using cardiovascular magnetic resonanceimaging and biochemical
markers. Circulation. 109:345–350. 2004. View Article : Google Scholar
|
10
|
Menasché P: The systemic factor: the
comparative roles of cardiopulmonary bypass and off-pump surgery in
the genesis of patient injury during and following cardiac surgery.
Ann Thorac Surg. 72:S2260–S2265; discussion S2265–S2266,
S2267–S2270. 2001.PubMed/NCBI
|
11
|
Tomic V, Russwurm S, Moller E, et al:
Transcriptomic and proteomic patterns of systemic inflammation in
on-pump and off-pump coronary artery bypass grafting. Circulation.
112:2912–2920. 2005.
|
12
|
Brown J, Hernandez F, Beaulieu P, Clough
R, Whited C, et al: Off-pump coronary artery bypass does not
influence biomarkers of brain injury, but does exacerbate the
systemic inflammatory response. J Clin Cell Immunik.
S2:0012011.
|
13
|
Frassdorf J, De Hert S and Schlack W:
Anaesthesia and myocardial ischaemia/reperfusion injury. Br J
Anaesth. 103:89–98. 2009. View Article : Google Scholar : PubMed/NCBI
|
14
|
Varadarajan SG, An J, Novalija E and Stowe
DF: Sevoflurane before or after ischemia improves contractile and
metabolic function while reducing myoplasmic Ca(2+)
loading in intact hearts. Anesthesiology. 96:125–133. 2002.
View Article : Google Scholar : PubMed/NCBI
|
15
|
Preckel B, Schlack W, Comfère T, Obal D,
Barthel H and Thämer V: Effects of enflurane, isoflurane,
sevoflurane and desflurane on reperfusion injury after regional
myocardial ischaemia in the rabbit heart in vivo. Br J Anaesth.
81:905–912. 1998. View Article : Google Scholar
|
16
|
Lin E and Symons JA: Volatile anaesthetic
myocardial protection: a review of the current literature. HSR Proc
Intensive Care Cardiovasc Anesth. 2:105–109. 2010.
|
17
|
Garcia C, Julier K, Bestmann L, et al:
Preconditioning with sevoflurane decreases PECAM-1 expression and
improves one-year cardiovascular outcome in coronary artery bypass
graft surgery. Br J Anaesth. 94:159–165. 2005.PubMed/NCBI
|
18
|
Kawamura T, Kadosaki M, Nara N, et al:
Effects of sevoflurane on cytokine balance in patients undergoing
coronary artery bypass graft surgery. J Cardiothorac Vasc Anesth.
20:503–508. 2006. View Article : Google Scholar : PubMed/NCBI
|
19
|
Huang Z, Zhong X, Irwin MG, et al: Synergy
of isoflurane preconditioning and propofol postconditioning reduces
myocardial reperfusion injury in patients. Clin Sci (Lond).
121:57–69. 2011. View Article : Google Scholar
|
20
|
Corcoran TB, Engel A, Sakamoto H, O’shea
A, O’callaghan-Enright S and Shorten GD: The effects of propofol on
neutrophil function, lipid peroxidation and inflammatory response
during elective coronary artery bypass grafting in patients with
impaired ventricular function. Br J Anaesth. 97:825–831. 2006.
View Article : Google Scholar
|
21
|
Sun HY, Xue FS, Xu YC, et al: Propofol
improves cardiac functional recovery after ischemia-reperfusion by
upregulating nitric oxide synthase activity in the isolated rat
hearts. Chin Med J (Engl). 122:3048–3054. 2009.
|
22
|
Wang HY, Wang GL, Yu YH and Wang Y: The
role of phosphoinositide-3-kinase/Akt pathway in propofol-induced
postconditioning against focal cerebral ischemia-reperfusion injury
in rats. Brain Res. 1297:177–184. 2009. View Article : Google Scholar
|
23
|
Kottenberg E, Thielmann M, Bergmann L, et
al: Protection by remote ischemic preconditioning during coronary
artery bypass graft surgery with isoflurane but not propofol - a
clinical trial. Acta Anaesthesiol Scand. 56:30–38. 2012. View Article : Google Scholar
|
24
|
Zaugg M, Lucchinetti E, Spahn DR, Pasch T,
Garcia C and Schaub MC: Differential effects of anesthetics on
mitochondrial K(ATP) channel activity and cardiomyocyte protection.
Anesthesiology. 97:15–23. 2002. View Article : Google Scholar : PubMed/NCBI
|
25
|
Müllenheim J, Ebel D, Frässdorf J, Preckel
B, Thämer V and Schlack W: Isoflurane preconditions myocardium
against infarction via release of free radicals. Anesthesiology.
96:934–940. 2002.PubMed/NCBI
|
26
|
Heusch G, Boengler K and Schulz R:
Cardioprotection: nitric oxide, protein kinases and mitochondria.
Circulation. 118:1915–1919. 2008. View Article : Google Scholar : PubMed/NCBI
|
27
|
He W, Zhang FJ, Wang SP, Chen G, Chen CC
and Yan M: Postconditioning of sevoflurane and propofol is
associated with mitochondrial permeability transition pore. J
Zhejiang Univ Sci B. 9:100–108. 2008. View Article : Google Scholar : PubMed/NCBI
|
28
|
Lucchinetti E, Hofer C, Bestmann L, et al:
Gene regulatory control of myocardial energy metabolism predicts
postoperative cardiac function in patients undergoing off-pump
coronary artery bypass graft surgery: inhalational versus
intravenous anesthetics. Anesthesiology. 106:444–457. 2007.
View Article : Google Scholar
|
29
|
Fujita A, Sato JR, de Rodrigues LO,
Ferreira CE and Sogayar MC: Evaluating different methods of
microarray data normalization. BMC Bioinformatic. 7:4692006.
View Article : Google Scholar : PubMed/NCBI
|
30
|
Smyth GK: Limma: linear models for
microarray data. Bioinformatics and Computational Biology Solutions
using R and Bioconductor. Gentleman R, Carey V, Dudoit S, Irazarry
R and Huber W: Springer; New York, NY: pp. 397–420. 2005,
View Article : Google Scholar
|
31
|
Benjamini Y and Hochberg Y: Controlling
the false discovery rate: a practical and powerful approach to
multiple testing. J R Stat Soc B. 1:289–300. 1995.
|
32
|
Huang Da W, Sherman BT and Lempicki RA:
Systematic and integrative analysis of large gene lists using DAVID
Bioinformatics Resources. Nature Protoc. 4:44–57. 2009.PubMed/NCBI
|
33
|
Tatusov RL, Natale DA, Garkavtsev IV, et
al: The COG database: new developments in phylogenetic
classification of proteins from complete genomes. Nucleic Acids
Res. 29:22–28. 2001. View Article : Google Scholar : PubMed/NCBI
|
34
|
Altschul SF, Gish W, Miller W, Myers EW
and Lipman DJ: Basic local alignment search tool. J Mol Biol.
215:403–410. 1990. View Article : Google Scholar
|
35
|
Zhang B, Kirov S and Snoddy J: WebGestalt:
an integrated system for exploring gene sets in various biological
contexts. Nucleic Acids Res. 33:W741–W748. 2005. View Article : Google Scholar : PubMed/NCBI
|
36
|
Shi G, Zhang L and Jiang T: MSOAR 2.0:
Incorporating tandem duplications into ortholog assignment based on
genome rearrangement. BMC Bioinformatics. 11:102010. View Article : Google Scholar : PubMed/NCBI
|
37
|
Malik V, Kale SC, Chowdhury UK,
Ramakrishnan L, Chauhan S and Kiran U: Myocardial injury in
coronary artery bypass grafting: On-pump versus off-pump comparison
by measuring heart-type fatty-acid-binding protein release. Tex
Heart Inst J. 33:321–327. 2006.
|
38
|
Mathur S, Farhangkhgoee P and Karmazyn M:
Cardioprotective effects of propofol and sevoflurane in ischemic
and reperfused rat hearts: role of K(ATP) channels and interaction
with the sodium-hydrogen exchange inhibitor HOE 642 (cariporide).
Anesthesiology. 91:1349–1360. 1999. View Article : Google Scholar : PubMed/NCBI
|
39
|
Nobori K, Ito H, Tamamori-Adachi M, et al:
ATF3 inhibits doxorubicin-induced apoptosis in cardiac myocytes: a
novel cardioprotective role of ATF3. J Mol Cell Cardiol.
34:1387–1397. 2002. View Article : Google Scholar : PubMed/NCBI
|
40
|
Seijffers R, Allchorne AJ and Woolf CJ:
The transcription factor ATF-3 promotes neurite outgrowth. Mol Cell
Neurosci. 32:143–154. 2006. View Article : Google Scholar : PubMed/NCBI
|
41
|
Francis JS, Dragunow M and During MJ: Over
expression of ATF-3 protects rat hippocampal neurons from in vivo
injection of kainic acid. Brain Res Mol Brain Res. 124:199–203.
2004. View Article : Google Scholar : PubMed/NCBI
|
42
|
Chu HM, Tan Y, Kobierski LA, Balsam LB and
Comb MJ: Activating transcription factor-3 stimulates 3′,5′-cyclic
adenosine monophosphate-dependent gene expression. Mol Endocrinol.
8:59–68. 1994.
|
43
|
Weitzman JB, Fiette L, Matsuo K and Yaniv
M: JunD protects cells from p53-dependent senescence and apoptosis.
Mol Cell. 6:1109–1119. 2000. View Article : Google Scholar : PubMed/NCBI
|
44
|
Bergman MR, Cheng S, Honbo N, Piacentini
L, Karliner JS and Lovett DH: A functional activating protein 1
(AP-1) site regulates matrix metalloproteinase 2 (MMP-2)
transcription by cardiac cells through interactions with JunB-Fra1
and JunB-FosB heterodimers. Biochem J. 369:485–496. 2003.
View Article : Google Scholar
|
45
|
Alfonso-Jaume MA, Bergman MR, Mahimkar R,
et al: Cardiac ischemia-reperfusion injury induces matrix
metalloproteinase-2 expression through the AP-1 components FosB and
JunB. Am J Physiol Heart Circ Physiol. 291:H1838–H1846. 2006.
View Article : Google Scholar
|
46
|
Kumar S, Boehm J and Lee JC: p38 MAP
kinases: key signalling molecules as therapeutic targets for
inflammatory diseases. Nat Rev Drug Discov. 2:717–726. 2003.
View Article : Google Scholar : PubMed/NCBI
|
47
|
Muslin AJ: MAPK signalling in
cardiovascular health and disease: molecular mechanisms and
therapeutic targets. Clin Sci (Lond). 115:203–218. 2008. View Article : Google Scholar : PubMed/NCBI
|
48
|
Wang Y, Huang S, Sah VP, et al: Cardiac
muscle cell hypertrophy and apoptosis induced by distinct members
of the p38 mitogen-activated protein kinase family. J Biol Chem.
273:2161–2168. 1998. View Article : Google Scholar : PubMed/NCBI
|
49
|
Boonyasrisawat W, Eberle D, Bacci S, et
al: Tag polymorphisms at the A20 (TNFAIP3) locus are associated
with lower gene expression and increased risk of coronary artery
disease in type 2 diabetes. Diabetes. 56:499–505. 2007. View Article : Google Scholar : PubMed/NCBI
|
50
|
Xue H, Wang SX, Wang XJ, et al: Variants
of tumor necrosis factor-induced protein 3 gene are associated with
left ventricular hypertrophy in hypertensive patients. Chin Med J
(Engl). 124:1498–1503. 2011.
|
51
|
De Keulenaer GW, Wang Y, Feng Y, et al:
Identification of IEX-1 as a biomechanically controlled nuclear
factor-kappaB target gene that inhibits cardiomyocyte hypertrophy.
Circ Res. 90:690–696. 2002.
|
52
|
Hirotani S, Otsu K, Nishida K, et al:
Involvement of nuclear factor-kappaB and apoptosis
signal-regulating kinase 1 in G-protein-coupled receptor
agonist-induced cardiomyocyte hypertrophy. Circulation.
105:509–515. 2002. View Article : Google Scholar
|