1
|
Ozaki K, Sato H, Inoue K, et al: SNPs in
BRAP associated with risk of myocardial infarction in Asian
populations. Nat Genet. 41:329–333. 2009. View Article : Google Scholar : PubMed/NCBI
|
2
|
Chacko SM, Khan M, Kuppusamy ML, et al:
Myocardial oxygenation and functional recovery in infarct rat
hearts transplanted with mesenchymal stem cells. Am J Physiol Heart
Circ Physiol. 296:H1263–H1273. 2009. View Article : Google Scholar : PubMed/NCBI
|
3
|
Cohen-Armon M: PARP-1 activation in the
ERK signaling pathway. Trends Pharmacol Sci. 28:556–560. 2007.
View Article : Google Scholar : PubMed/NCBI
|
4
|
Hassa PO, Haenni SS, Elser M and Hottiger
MO: Nuclear ADP-ribosylation reactions in mammalian cells: where
are we today and where are we going? Microbiol Mol Biol Rev.
70:789–829. 2006. View Article : Google Scholar : PubMed/NCBI
|
5
|
Szabó C, Zingarelli B, O’Connor M and
Salzman AL: DNA strand breakage, activation of poly (ADP-ribose)
synthetase, and cellular energy depletion are involved in the
cytotoxicity of macrophages and smooth muscle cells exposed to
peroxynitrite. Proc Natl Acad Sci USA. 93:1753–1758. 1996.
View Article : Google Scholar : PubMed/NCBI
|
6
|
Ullrich O, Diestel A, Eyüpoglu IY and
Nitsch R: Regulation of microglial expression of integrins by
poly(ADP-ribose) polymerase-1. Nat Cell Biol. 3:1035–1042. 2001.
View Article : Google Scholar
|
7
|
Sharp C, Warren A, Oshima T, Williams L,
Li JH and Alexander JS: Poly ADP ribose-polymerase inhibitors
prevent the upregulation of ICAM-1 and E-selectin in response to
Th1 cytokine stimulation. Inflammation. 25:157–163. 2001.
View Article : Google Scholar : PubMed/NCBI
|
8
|
Hassa PO and Hottiger MO: The functional
role of poly (ADP-ribose)polymerase 1 as novel coactivator of
NF-kappaB in inflammatory disorders. Cell Mol Life Sci.
59:1534–1553. 2002. View Article : Google Scholar : PubMed/NCBI
|
9
|
Carrillo A, Monreal Y, Ramírez P, et al:
Transcription regulation of TNF-alpha-early response genes by
poly(ADP-ribose) polymerase-1 in murine heart endothelial cells.
Nucleic Acids Res. 32:757–766. 2004. View Article : Google Scholar : PubMed/NCBI
|
10
|
Szabó C, Zanchi A, Komjáti K, et al:
Poly(ADP-Ribose) polymerase is activated in subjects at risk of
developing type 2 diabetes and is associated with impaired vascular
reactivity. Circulation. 106:2680–2686. 2002. View Article : Google Scholar : PubMed/NCBI
|
11
|
Pacher P, Beckman JS and Liaudet L: Nitric
oxide and peroxynitrite in health and disease. Physiol Rev.
87:315–424. 2007. View Article : Google Scholar : PubMed/NCBI
|
12
|
Pacher P and Szabó C: Role of
poly(ADP-ribose) polymerase-1 activation in the pathogenesis of
diabetic complications: endothelial dysfunction, as a common
underlying theme. Antioxid Redox Signal. 7:1568–1580. 2005.
View Article : Google Scholar : PubMed/NCBI
|
13
|
Martinet W, Knaapen MW, De Meyer GR,
Herman AG and Kockx MM: Elevated levels of oxidative DNA damage and
DNA repair enzymes in human atherosclerotic plaques. Circulation.
106:927–932. 2002. View Article : Google Scholar : PubMed/NCBI
|
14
|
Szabó C: Pharmacological inhibition of
poly(ADP-ribose) polymerase in cardiovascular disorders: future
directions. Curr Vasc Pharmacol. 3:301–303. 2005. View Article : Google Scholar : PubMed/NCBI
|
15
|
Szabó C, Ischiropoulos H and Radi R:
Peroxynitrite: biochemistry, pathophysiology and development of
therapeutics. Nat Rev Drug Discov. 6:662–680. 2007. View Article : Google Scholar : PubMed/NCBI
|
16
|
Ahmet I, Lakatta EG and Talan MI:
Pharmacological stimulation of beta2-adrenergic receptors (beta2AR)
enhances therapeutic effectiveness of beta1AR blockade in rodent
dilated ischemic cardiomyopathy. Heart Fail Rev. 10:289–296. 2005.
View Article : Google Scholar
|
17
|
Matsuhisa S, Otani H, Okazaki T, et al:
Angiotensin II type 1 receptor blocker preserves tolerance to
ischemia-reperfusion injury in Dahl salt-sensitive rat heart. Am J
Physiol Heart Circ Physiol. 294:H2473–H2479. 2008. View Article : Google Scholar : PubMed/NCBI
|
18
|
Wang Y, Zhang G, Hou Y, et al:
Transplantation of microencapsulated Schwann cells and mesenchymal
stem cells augment angiogenesis and improve heart function. Mol
Cell Biochem. 366:139–147. 2012. View Article : Google Scholar : PubMed/NCBI
|
19
|
Khan M, Meduru S, Mohan IK, et al:
Hyperbaric oxygenation enhances transplanted cell graft and
functional recovery in the infarct heart. J Mol Cell Cardiol.
47:275–287. 2009. View Article : Google Scholar : PubMed/NCBI
|
20
|
Ahmet I, Tae HJ, Juhaszova M, et al: A
small nonerythropoietic helix B surface peptide based upon
erythropoietin structure is cardioprotective against ischemic
myocardial damage. Mol Med. 17:194–200. 2011. View Article : Google Scholar :
|
21
|
Pacher P and Szabó C: Role of
poly(ADP-ribose) polymerase 1 (PARP-1) in cardiovascular diseases:
the therapeutic potential of PARP inhibitors. Cardiovasc Drug Rev.
25:235–260. 2007. View Article : Google Scholar : PubMed/NCBI
|
22
|
Levrand S, Vannay-Bouchiche C, Pesse B, et
al: Peroxynitrite is a major trigger of cardiomyocyte apoptosis in
vitro and in vivo. Free Radic Biol Med. 41:886–895. 2006.
View Article : Google Scholar : PubMed/NCBI
|
23
|
Virag L and Szabó C: The therapeutic
potential of poly(ADP-ribose) polymerase inhibitors. Pharmacol Rev.
54:375–429. 2002. View Article : Google Scholar : PubMed/NCBI
|
24
|
Kumar D and Jugdutt BI: Apoptosis and
oxidants in the heart. J Lab Clin Med. 142:288–297. 2003.
View Article : Google Scholar : PubMed/NCBI
|
25
|
Fiorillo C, Ponziani V, Giannini L, et al:
Protective effects of the PARP-1 inhibitor PJ34 in
hypoxic-reoxygenated cardiomyoblasts. Cell Mol Life Sci.
63:3061–3071. 2006. View Article : Google Scholar : PubMed/NCBI
|
26
|
Szabó C: Poly(ADP-ribose) polymerase
activation by reactive nitrogen species - relevance for the
pathogenesis of inflammation. Nitric Oxide. 14:169–179. 2006.
View Article : Google Scholar
|
27
|
Sarkar D and Fisher PB: Molecular
mechanisms of aging-associated inflammation. Cancer Lett.
236:13–23. 2006. View Article : Google Scholar
|
28
|
Gilson WD, Epstein FH, Yang Z, et al:
Borderzone contractile dysfunction is transiently attenuated and
left ventricular structural remodeling is markedly reduced
following reperfused myocardial infarction in inducible nitric
oxide synthase knockout mice. J Am Coll Cardiol. 50:1799–807. 2007.
View Article : Google Scholar : PubMed/NCBI
|
29
|
Liu YH, Carretero OA, Cingolani OH, et al:
Role of inducible nitric oxide synthase in cardiac function and
remodeling in mice with heart failure due to myocardial infarction.
Am J Physiol Heart Circ Physiol. 289:H2616–23. 2005. View Article : Google Scholar : PubMed/NCBI
|
30
|
Mukhopadhyay P, Rajesh M, Bátkai S, et al:
Role of superoxide, nitric oxide, and peroxynitrite in
doxorubicin-induced cell death in vivo and in vitro. Am J Physiol
Heart Circ Physiol. 296:H1466–H1483. 2009. View Article : Google Scholar : PubMed/NCBI
|
31
|
van Wijk SJ and Hageman GJ:
Poly(ADP-ribose) polymerase-1 mediated caspase-independent cell
death after ischemia/reperfusion. Free Radic Biol Med. 39:81–90.
2005. View Article : Google Scholar : PubMed/NCBI
|
32
|
Zhang Y, Zhang X, Park TS and Gidday JM:
Cerebral endothelial cell apoptosis after ischemia-reperfusion:
role of PARP activation and AIF translocation. J Cereb Blood Flow
Metab. 25:868–877. 2005. View Article : Google Scholar : PubMed/NCBI
|