1
|
Iso H: Changes in coronary heart disease
risk among Japanese. Circulation. 118:2725–2729. 2008. View Article : Google Scholar : PubMed/NCBI
|
2
|
Yeh RW and Go AS: Rethinking the
epidemiology of acute myocardial infarction: challenges and
opportunities. Arch Intern Med. 170:759–764. 2010. View Article : Google Scholar : PubMed/NCBI
|
3
|
Yusuf S, Hawken S, Ounpuu S, et al: Effect
of potentially modifiable risk factors associated with myocardial
infarction in 52 countries (the INTERHEART study): case-control
study. Lancet. 364:937–952. 2004. View Article : Google Scholar : PubMed/NCBI
|
4
|
Keith DS, Nichols GA, Gullion CM, et al:
Longitudinal follow-up and outcomes among a population with chronic
kidney disease in a large managed care organization. Arch Intern
Med. 164:659–663. 2004. View Article : Google Scholar : PubMed/NCBI
|
5
|
Chan L and Boerwinkle E: Gene-environment
interactions and gene therapy in atherosclerosis. Cardiol Rev.
2:130–137. 1994. View Article : Google Scholar
|
6
|
Yamada Y, Izawa H, Ichihara S, et al:
Prediction of the risk of myocardial infarction from polymorphisms
in candidate genes. N Engl J Med. 347:1916–1923. 2002. View Article : Google Scholar : PubMed/NCBI
|
7
|
Arnett DK, Baird AE, Barkley RA, et al;
American Heart Association Council on Epidemiology and Prevention;
American Heart Association Stroke Council; Functional Genomics and
Translational Biology Interdisciplinary Working Group. Relevance of
genetics and genomics for prevention and treatment of
cardiovascular disease: a scientific statement from the American
Heart Association Council on Epidemiology and Prevention, the
Stroke Council, and the Functional Genomics and Translational
Biology Interdisciplinary Working Group. Circulation.
115:2878–2901. 2007.
|
8
|
Wellcome Trust Case Control Consortium.
Genome-wide association study of 14,000 cases of seven common
diseases and 3,000 shared controls. Nature. 447:661–678. 2007.
View Article : Google Scholar : PubMed/NCBI
|
9
|
Helgadottir A, Thorleifsson G, Manolescu
A, et al: A common variant on chromosome 9p21 affects the risk of
myocardial infarction. Science. 316:1491–1493. 2007. View Article : Google Scholar : PubMed/NCBI
|
10
|
Samani NJ, Erdmann J, Hall AS, et al;
WTCCC and the Cardiogenics Consortium. Genomewide association
analysis of coronary artery disease. N Engl J Med. 357:443–453.
2007. View Article : Google Scholar : PubMed/NCBI
|
11
|
Coronary Artery Disease (C4D) Genetics
Consortium. A genome-wide association study in Europeans and South
Asians identifies five new loci for coronary artery disease. Nat
Genet. 43:339–344. 2011. View
Article : Google Scholar : PubMed/NCBI
|
12
|
Yamada Y, Nishida T, Ichihara S, et al:
Identification of chromosome 3q28 and ALPK1 as susceptibility loci
for chronic kidney disease in Japanese individuals by a genome-wide
association study. J Med Genet. 50:410–418. 2013. View Article : Google Scholar : PubMed/NCBI
|
13
|
Itoh Y, Mizuki N, Shimada T, et al:
High-throughput DNA typing of HLA-A, -B, -C, and -DRB1 loci by a
PCR-SSOP-Luminex method in the Japanese population. Immunogenetics.
57:717–729. 2005. View Article : Google Scholar : PubMed/NCBI
|
14
|
Ryazanov AG, Pavur KS, Dorovkov MV, et al:
Alpha-kinases: a new class of protein kinases with a novel
catalytic domain. Curr Biol. 9:R43–R45. 1999. View Article : Google Scholar : PubMed/NCBI
|
15
|
Middelbeek J, Clark K, Venselaar H, et al:
The alpha-kinase family: an exceptional branch on the protein
kinase tree. Cell Mol Life Sci. 67:875–890. 2010. View Article : Google Scholar : PubMed/NCBI
|
16
|
Heine M, Cramm-Behrens CI, Ansari A, et
al: Alpha-kinase 1, a new component in apical protein transport. J
Biol Chem. 280:25637–25643. 2005. View Article : Google Scholar : PubMed/NCBI
|
17
|
Wang SJ, Tu HP, Ko AM, et al: Lymphocyte
α-kinase is a gout-susceptible gene involved in monosodium urate
monohydrate-induced inflammatory responses. J Mol Med (Berl).
89:1241–1251. 2011.
|
18
|
Hansson GK: Inflammation, atherosclerosis,
and coronary artery disease. N Engl J Med. 352:1685–1695. 2005.
View Article : Google Scholar : PubMed/NCBI
|
19
|
Libby P, Ridker PM, Hansson GK, et al:
Inflammation in atherosclerosis: from pathophysiology to practice.
J Am Coll Cardiol. 54:2129–2138. 2009. View Article : Google Scholar : PubMed/NCBI
|
20
|
Newman DJ, Thakkar H, Edwards RG, et al:
Serum cystatin C measured by automated immunoassay: a more
sensitive marker of changes in GFR than serum creatinine. Kidney
Int. 47:312–318. 1995. View Article : Google Scholar : PubMed/NCBI
|
21
|
Singh D, Whooley MA, Ix JH, Ali S and
Shlipak MG: Association of cystatin C and estimated GFR with
inflammatory biomarkers: the Heart and Soul Study. Nephrol Dial
Transplant. 22:1087–1092. 2007. View Article : Google Scholar : PubMed/NCBI
|
22
|
Shlipak MG, Katz R, Cushman M, et al:
Cystatin-C and inflammatory markers in the ambulatory elderly. Am J
Med. 118:14162005. View Article : Google Scholar : PubMed/NCBI
|
23
|
Qing X, Furong W, Yunxia L, et al:
Cystatin C and asymptomatic coronary artery disease in patients
with metabolic syndrome and normal glomerular filtration rate.
Cardiovasc Diabetol. 11:1082012. View Article : Google Scholar : PubMed/NCBI
|
24
|
Koenig W, Twardella D, Brenner H, et al:
Plasma concentrations of cystatin C in patients with coronary heart
disease and risk for secondary cardiovascular events: more than
simply a marker of glomerular filtration rate. Clin Chem.
51:321–327. 2005. View Article : Google Scholar : PubMed/NCBI
|