|
1
|
Pang MYC and Eng JJ: Muscle strength is a
determinant of bone mineral content in the hemiparetic upper
extremity: Implications for stroke rehabilitation. Bone.
37:103–111. 2005. View Article : Google Scholar : PubMed/NCBI
|
|
2
|
Iwamoto J, Takeda T and Ichimura S:
Relationships between physical activity and metacarpal cortical
bone mass and bone resorption in hemiplegic patients. J Orthop Sci.
6:227–233. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
3
|
Sahin L, Ozoran K, Gündüz OH, Uçan H and
Yücel M: Bone mineral density in patients with stroke. Am J Phys
Med Rehabil. 80:592–596. 2001. View Article : Google Scholar : PubMed/NCBI
|
|
4
|
Nielsen S and Pedersen BK: Skeletal muscle
as an immunogenic organ. Curr Opin Pharmacol. 8:346–351. 2008.
View Article : Google Scholar : PubMed/NCBI
|
|
5
|
Engler D: Hypothesis: Musculin is a
hormone secreted by skeletal muscle, the body's largest endocrine
organ. Evidence for actions on the endocrine pancreas to restrain
the beta-cell mass and to inhibit insulin secretion and on the
hypothalamus to co-ordinate the neuroendocrine and appetite
responses to exercise. Acta Biomed. 78(Suppl 1): 156–206.
2007.PubMed/NCBI
|
|
6
|
Ma K, Mallidis C, Bhasin S, Mahabadi V,
Artaza J, et al: Glucocorticoid-induced skeletal muscle atrophy is
associated with upregulation of myostatin gene expression. Am J
Physiol Endocrinol Metab. 285:E363–E371. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
7
|
Elkasrawy MN and Hamrick MW: Myostatin
(GDF-8) as a key factor linking muscle mass and bone structure. J
Musculoskelet Neuronal Interact. 10:56–63. 2010.PubMed/NCBI
|
|
8
|
Hamrick MW, Samaddar T, Pennington C and
McCormick J: Increased muscle mass with myostatin deficiency
improves gains in bone strength with exercise. J Bone Miner Res.
21:477–483. 2006. View Article : Google Scholar : PubMed/NCBI
|
|
9
|
West CA, Arnett TR and Farrow SM:
Expression of insulin-like growth factor I (IGF-I) mRNA variants in
rat bone. Bone. 19:41–46. 1996. View Article : Google Scholar : PubMed/NCBI
|
|
10
|
Wei CN, Ohira Y, Tanaka T, Yonemitsu H and
Ueda A: Does electrical stimulation of the sciatic nerve prevent
suspension-induced changes in rat hindlimb bones? Jpn J Physiol.
48:33–37. 1998. View Article : Google Scholar : PubMed/NCBI
|
|
11
|
Roth SM, Martel GF, Ferrell RE, Metter EJ,
Hurley BF and Rogers MA: Myostatin gene expression is reduced in
humans with heavy-resistance strength training: A brief
communication. Exp Biol Med (Maywood). 228:706–709. 2003.PubMed/NCBI
|
|
12
|
Hill M and Goldspink G: Expression and
splicing of the insulin-like growth factor gene in rodent muscle is
associated with muscle satellite (stem) cell activation following
local tissue damage. J Physiol. 549:409–418. 2003. View Article : Google Scholar : PubMed/NCBI
|
|
13
|
Riddoch-Contreras J, Yang SY, Dick JR,
Goldspink G, Orrell RW and Greensmith L: Mechano-growth factor, an
IGF-I splice variant, rescues motoneurons and improves muscle
function in SOD1(G93A) mice. Exp Neurol. 215:281–289. 2009.
View Article : Google Scholar : PubMed/NCBI
|
|
14
|
Hameed M, Orrell RW, Cobbold M, et al:
Expression of IGF-I splice variants in young and old human skeletal
muscle after high resistance exercise. J Physiol. 547:247–254.
2003. View Article : Google Scholar : PubMed/NCBI
|
|
15
|
Bamman MM, Shipp JR, Jiang J, Gower BA,
Hunter GR, Goodman A, et al: Mechanical load increases muscle IGF-I
and androgen receptor mRNA concentrations in humans. Am J Physiol
Endocrinol Metab. 280:E383–E390. 2001.PubMed/NCBI
|
|
16
|
Owino V, Yang SY and Goldspink G:
Age-related loss of skeletal muscle function and the inability to
express the autocrine form of insulin-like growth factor-1 (MGF) in
response to mechanical overload. FEBS Lett. 505:259–263. 2001.
View Article : Google Scholar : PubMed/NCBI
|
