1
|
Hodson EM, Knight JF, Willis NS and Craig
JC: Corticosteroid therapy for nephrotic syndrome in children.
Cochrane Database Syst Rev. 2:CD0015332004.
|
2
|
Hardwicke J, Soothill JF, Squire JR and
Holti G: Nephrotic syndrome with pollen hypersensitivity. Lancet.
1:500–502. 1959. View Article : Google Scholar : PubMed/NCBI
|
3
|
Mack M and Rosenkranz AR: Basophils and
mast cells in renal injury. Kidney Int. 76:1142–1147. 2009.
View Article : Google Scholar : PubMed/NCBI
|
4
|
Lagrue G and Laurent J: Allergy and lipoid
nephrosis. Adv Nephrol Necker Hosp. 12:151–175. 1983.PubMed/NCBI
|
5
|
Reeves WG, Cameron JS, Johansson SG, et
al: Seasonal nephrotic syndrome. Description and immunological
findings. Clin Allergy. 5:121–137. 1975. View Article : Google Scholar : PubMed/NCBI
|
6
|
Laqrue G and Laurent J: Nephrotic syndrome
and hypersensitivity to pollens (author’s transl). Nouv Presse Med.
11:1479–1481. 1982.(In French).
|
7
|
Rytand DA: Onset of the nephrotic syndrome
during a reaction to bee sting. Stanford Med Bull. 13:224–233.
1955.PubMed/NCBI
|
8
|
Schulte-Wisserman H, Görtz W and Straub E:
IgE in patients with glomerulonephritis and minimal-change
nephrotic syndrome. Eur J Pediatr. 131:105–111. 1979. View Article : Google Scholar : PubMed/NCBI
|
9
|
Chan MK, Chan KW and Jones B:
Immunoglobulins (IgG, IgA, IgM, IgE) and complement components (C3,
C4) in nephrotic syndrome due to minimal change and other forms of
glomerulonephritis, a clue for steroid therapy? Nephron.
47:125–130. 1987. View Article : Google Scholar : PubMed/NCBI
|
10
|
Shu KH, Lian JD, Yang YF, et al: Serum IgE
in primary glomerular diseases and its clinical significance.
Nephron. 49:24–28. 1988. View Article : Google Scholar : PubMed/NCBI
|
11
|
Reeves WG, Cameron JS and Ogg CS: IgE and
the nephrotic syndrome. Lancet. 1:1299–1300. 1971. View Article : Google Scholar : PubMed/NCBI
|
12
|
Yokoyama H, Kida H, Tani Y, et al:
Immunodynamics of minimal change nephrotic syndrome in adults T and
B lymphocyte subsets and serum immunoglobulin levels. Clin Exp
Immunol. 61:601–607. 1985.PubMed/NCBI
|
13
|
Kang J, Bai KM, Wang BL, et al: Increased
production of interleukin 4 in children with simple idiopathic
nephrotic syndrome. Chin Med J (Engl). 107:347–350. 1994.PubMed/NCBI
|
14
|
Cho BS, Yoon SR, Jang JY, et al:
Up-regulation of interleukin-4 and CD23/FcepsilonRII in minimal
change nephrotic syndrome. Pediatr Nephrol. 13:199–204. 1999.
View Article : Google Scholar : PubMed/NCBI
|
15
|
Lama G, Luong I, Tirino G, et al:
T-lymphocyte populations and cytokines in childhood nephrotic
syndrome. Am J Kidney Dis. 39:958–965. 2002. View Article : Google Scholar : PubMed/NCBI
|
16
|
Gong WK, Cheung W and Yap HK: Minimal
change nephrotic syndrome - a complex genetic disorder. Ann Acad
Med Singapore. 29:351–356. 2000.PubMed/NCBI
|
17
|
Falcone FH, Zillikens D and Gibbs BF: The
21st century renaissance of the basophil? Current insights into its
role in allergic responses and innate immunity. Exp Dermatol.
15:855–864. 2006. View Article : Google Scholar : PubMed/NCBI
|
18
|
Shalhoub RJ: Pathogenesis of lipoid
nephrosis: a disorder of T-cell function. Lancet. 2:556–560. 1974.
View Article : Google Scholar : PubMed/NCBI
|
19
|
Okada H, Kuhn C, Feillet H and Bach JF:
The ‘hygiene hypothesis’ for autoimmune and allergic diseases: an
update. Clin Exp Immunol. 160:1–9. 2010.
|
20
|
Hurtado A and Johnson RJ: Hygiene
hypothesis and prevalence of glomerulonephritis. Kidney Int Suppl.
97:S62–S67. 2005. View Article : Google Scholar : PubMed/NCBI
|
21
|
Masutani K, Taniguchi M, Nakashima H,
Yotsueda H, et al: Up-regulated interleukin-4 production by
peripheral T-helper cells in idiopathic membranous nephropathy.
Nephrol Dial Transplant. 19:580–586. 2004. View Article : Google Scholar : PubMed/NCBI
|
22
|
Shimoyama H, Nakajima M, Naka H, Maruhashi
Y, et al: Up-regulation of interleukin-2 mRNA in children with
idiopathic nephrotic syndrome. Pediatr Nephrol. 19:1115–1121. 2004.
View Article : Google Scholar : PubMed/NCBI
|
23
|
Yap HK, Cheung W, Murugasu B, et al: Th1
and Th2 cytokine mRNA profiles in childhood nephrotic syndrome:
evidence for increased IL-13 mRNA expression in relapse. J Am Soc
Nephrol. 10:529–537. 1999.PubMed/NCBI
|
24
|
Lama G, Luongo I, Tirino G, Borriello A,
et al: T-lymphocyte populations and cytokines in childhood
nephrotic syndrome. Am J Kidney Dis. 39:958–965. 2002. View Article : Google Scholar : PubMed/NCBI
|
25
|
Koyama A, Fujisaki M, Kobayashi M, et al:
A glomerular permeability factor produced by human T cell
hybridomas. Kidney Int. 40:453–460. 1991. View Article : Google Scholar : PubMed/NCBI
|
26
|
Adrogue HE, Borillo J, Torres L, et al:
Coincident activation of Th2 T cells with onset of the disease and
differential expression of GRO-gamma in peripheral blood leukocytes
in minimal change disease. Am J Nephrol. 27:253–261. 2007.
View Article : Google Scholar : PubMed/NCBI
|
27
|
Cho MH, Lee HS, Choe BH, et al:
Interleukin-8 and tumor necrosis factor-alpha are increased in
minimal change disease but do not alter albumin permeability. Am J
Nephrol. 23:260–266. 2003. View Article : Google Scholar : PubMed/NCBI
|
28
|
Parry RG, Gillespie KM, Parnham A, et al:
Interleukin-4 and interleukin-4 receptor polymorphisms in minimal
change nephropathy. Clin Sci (Lond). 96:665–668. 1999. View Article : Google Scholar : PubMed/NCBI
|
29
|
Abdel-Hafez M, Shimada M, Lee PY, et al:
Idiopathic nephrotic syndrome and atopy: is there a common link? Am
J Kidney Dis. 54:945–953. 2009. View Article : Google Scholar : PubMed/NCBI
|
30
|
Lai KW, Wei CL, Tan LK, Tan PH, et al:
Overexpression of interleukin-13 induces minimal-change-like
nephropathy in rats. J Am Soc Nephrol. 18:1476–1485. 2007.
View Article : Google Scholar : PubMed/NCBI
|
31
|
Hong YM, Liu ZH, Chen ZH, et al:
Interleukin-13 induced podocyte injury via a signal transducer and
activator of transcription-6-dependent way. Shen Zang Bing Yu Tou
Xi Shen Yi Zhi Za Zhi. 18:140–147. 2009.(In Chinese).
|
32
|
Tang TF, Hong YM, Chen ZH, et al:
Triptolide protects podocytes from IL-13 induced injury in vitro.
Yi Xue Yan Jiu Sheng Bao. 23:1140–1144. 2010.(In Chinese).
|
33
|
Pan QJ, Liu Y and Fu N: Progress of
research on basophils in allergic and immune reactions. Zhongguo
Mian Yi Xue Za Zhi. 25:671–673. 2009.(In Chinese).
|
34
|
Karasuyama H, Mukai K, Tsujimura Y and
Obata K: Newly discovered roles for basophils: a neglected minority
gains new respect. Nat Rev Immunol. 9:9–13. 2009. View Article : Google Scholar : PubMed/NCBI
|
35
|
Yoshimoto T, Yasuda K, Tanaka H, Nakahira
M, et al: Basophils contribute to T(H)2-IgE responses in vivo via
IL-4 production and presentation of peptide-MHC class II complexes
to CD4+ T cells. Nat Immunol. 10:706–712. 2009.
View Article : Google Scholar : PubMed/NCBI
|
36
|
Sokol CL, Barton GM, Farr AG and Medzhitov
R: A mechanism for the initiation of allergen-induced T helper type
2 responses. Nat Immunol. 9:310–318. 2008. View Article : Google Scholar : PubMed/NCBI
|
37
|
Kasaian MT, Clay MJ, Happ MP, Garman RD,
et al: IL-4 production by allergen-stimulated primary cultures:
identification of basophils as the major IL-4-producing cell type.
Int Immunol. 8:1287–1297. 1996. View Article : Google Scholar : PubMed/NCBI
|
38
|
Charles N, Watford WT, Ramos HL, et al:
Lyn kinase controls basophil GATA-3 transcription factor expression
and induction of Th2 cell differentiation. Immunity. 30:533–543.
2009. View Article : Google Scholar : PubMed/NCBI
|
39
|
Pirotzky E, Hieblot C, Benveniste J,
Laurent J, Lagrue G and Noirot C: Basophil sensitisation in
idiopathic nephrotic syndrome. Lancet. 1:358–361. 1982. View Article : Google Scholar : PubMed/NCBI
|
40
|
Laurent J, Lagrue G, Pirotzky E, Hirbec G,
Hieblot C and Benveniste J: Frequent positivity of the human
basophil degranulation test in idiopathic nephrotic syndromes
(minimal glomerular changes and segmental and focal
glomerulosclerosis). Nephrologie. 5:9–14. 1984.(In French).
|
41
|
Tan Y, Yang D, Fan J and Chen Y: Elevated
levels of immunoglobulin E may indicate steroid resistance or
relapse in adult primary nephrotic syndrome, especially in minimal
change nephrotic syndrome. J Int Med Res. 39:2307–2313. 2011.
View Article : Google Scholar
|
42
|
Fuke Y, Endo M, Ohsawa I, Satomura A, et
al: Implication of elevated serum IgE levels in minimal change
nephrotic syndrome. Nephron. 91:769–770. 2002. View Article : Google Scholar : PubMed/NCBI
|
43
|
Cairns SA, London RA and Mallick NP:
Circulating immune complexes in idiopathic glomerular disease.
Kidney Int. 21:507–512. 1982. View Article : Google Scholar : PubMed/NCBI
|
44
|
Charles N, Hardwick D, Daugas E, et al:
Basophils and the T helper 2 environment can promote the
development of lupus nephritis. Nat Med. 16:701–707. 2010.
View Article : Google Scholar : PubMed/NCBI
|
45
|
Buschor P, Eggel A, Zellweger F, Stadler
BM and Vogel M: Improved FcγRIIb targeting functionally translates
into enhanced inhibition of basophil activation. Int Arch Allergy
Immunol. 163:206–214. 2014.
|
46
|
MacGlashan D Jr: IgE receptor and signal
transduction in mast cells and basophils. Curr Opin Immunol.
20:717–723. 2008. View Article : Google Scholar : PubMed/NCBI
|
47
|
MacGlashan DW Jr: Self-termination/anergic
mechanisms in human basophils and mast cells. Int Arch Allergy
Immunol. 150:109–121. 2009. View Article : Google Scholar : PubMed/NCBI
|
48
|
Gilfillan AM, Peavy RD and Metcalfe DD:
Amplification mechanisms for the enhancement of antigen-mediated
mast cell activation. Immunol Res. 43:15–24. 2009. View Article : Google Scholar : PubMed/NCBI
|
49
|
Komiya A, Nagase H, Okugawa S, et al:
Expression and function of Toll-like receptors in human basophils.
Int Arch Allergy Immunol. 140(Suppl 1): 23–27. 2006. View Article : Google Scholar : PubMed/NCBI
|
50
|
Bieneman AP, Chichester KL, Chen YH and
Schroeder JT: Toll-like receptor 2 ligands activate human basophils
for both IgE-dependent and IgE-independent secretion. J Allergy
Clin Immunol. 115:295–301. 2005. View Article : Google Scholar : PubMed/NCBI
|
51
|
Takeda K and Akira S: Toll-like receptors
in innate immunity. Int Immunol. 17:1–14. 2005. View Article : Google Scholar
|
52
|
Creticos PS, Schroeder JT, Hamilton RG,
Balcer-Whaley SL, et al: Immunotherapy with a ragweed-Toll-like
receptor 9 agonist caccine for allergic rhinitis. N Engl J Med.
355:1445–1455. 2006. View Article : Google Scholar : PubMed/NCBI
|
53
|
Pan Q, Wang G, Ye L, Feng Y, Wang C and
Liu H: Toll-like receptor 9 is involved in the activation of
peripheral blood basophils of patients with systemic lupus
erythematosus. Clin Exp Allergy. 42:1821–1822. 2012.
|
54
|
Sabroe I, Jones EC, Usher LR, et al:
Toll-like receptor (TLR)2 and TLR4 in human peripheral blood
granulocytes: a critical role for monocytes in leukocyte
lipopolysaccharide responses. J Immunol. 168:4701–4710. 2002.
View Article : Google Scholar : PubMed/NCBI
|
55
|
Suurmond J, Stoop JN, Rivellese F, Bakker
AM, et al: Activation of human basophils by combined Toll-like
receptor- and FcɛRI-triggering can promote Th2 skewing of naive T
helper cells. Eur J Immunol. 44:386–396. 2014.PubMed/NCBI
|