1
|
Harris ED Jr: Rheumatoid arthritis.
Pathophysiology and implications for therapy. N Engl J Med.
322:1277–1289. 1990. View Article : Google Scholar : PubMed/NCBI
|
2
|
Schulze-Koops H and Kalden JR: The balance
of Th1/Th2 cytokines in rheumatoid arthritis. Best Pract Res Clin
Rheumatol. 15:677–691. 2001. View Article : Google Scholar : PubMed/NCBI
|
3
|
Woo P and Wedderburn LR: Juvenile chronic
arthritis. Lancet. 351:969–973. 1998. View Article : Google Scholar : PubMed/NCBI
|
4
|
Raziuddin S, Bahabri S, Al-Dalaan A, et
al: A mixed Th1/Th2 cell cytokine response predominates in systemic
onset juvenile rheumatoid arthritis: immunoregulatory IL-10
function. Clin Immunol Immunopathol. 86:192–198. 1998. View Article : Google Scholar : PubMed/NCBI
|
5
|
Zhang Q, Zhang H and Dong Z: A study on
immune response of Th1/Th2 in children with juvenile rheumatoid
arthritis. Acta Acad Med Qingdao Univ. 40:134–136. 2004.
|
6
|
Tang AT, Lau YL, Jones B, et al:
Cefadroxil reduces the production of IgE in a 3 year old asthmatic
with juvenile rheumatoid arthritis. Allergol Immunopathol (Madr).
21:131–135. 1993.PubMed/NCBI
|
7
|
Wedderburn LR, Robinson N, Patel A, et al:
Selective recruitment of polarized T cells expressing CCR5 and
CXCR3 to the inflamed joints of children with juvenile idiopathic
arthritis. Arthritis Rheum. 43:765–774. 2000. View Article : Google Scholar : PubMed/NCBI
|
8
|
Gerli R, Bistoni O, Russano A, et al: In
vivo activated T cells in rheumatoid synovitis. Analysis of Th1-
and Th2-type cytokine production at clonal level in different
stages of disease. Clin Exp Immunol. 129:549–555. 2002. View Article : Google Scholar : PubMed/NCBI
|
9
|
Raza K, Falciani F, Curnow SJ, et al:
Early rheumatoid arthritis is characterized by a distinct and
transient synovial fluid cytokine profile of T cell and stromal
cell origin. Arthritis Res Ther. 7:R784–R795. 2005. View Article : Google Scholar : PubMed/NCBI
|
10
|
Aarvak T, Chabaud M, Thoen J, et al:
Changes in the Th1 or Th2 cytokine dominance in the synovium of
rheumatoid arthritis (RA): a kinetic study of the Th subsets in one
unusual RA patient. Rheumatology (Oxford). 39:513–522. 2000.
View Article : Google Scholar : PubMed/NCBI
|
11
|
Chirumbolo S: State-of-the-art review
about basophil research in immunology and allergy: is the time
right to treat these cells with the respect they deserve? Blood
Transfus. 10:148–164. 2012.PubMed/NCBI
|
12
|
Falcone FH, Zillikens D and Gibbs BF: The
21st century renaissance of the basophil? Current insights into its
role in allergic responses and innate immunity. Exp Dermatol.
15:855–864. 2006. View Article : Google Scholar : PubMed/NCBI
|
13
|
Karasuyama H, Mukai K, Tsujimura Y and
Obata K: Newly discovered roles for basophils: a neglected minority
gains new respect. Nat Rev Immunol. 9:9–13. 2009. View Article : Google Scholar : PubMed/NCBI
|
14
|
Min B: Basophils induce Th2 immunity: Is
this final answer? Virulence. 1:399–401. 2010. View Article : Google Scholar : PubMed/NCBI
|
15
|
Sokol CL, Barton GM, Farr AG and Medzhitov
R: A mechanism for the initiation of allergen-induced T helper type
2 responses. Nat Immunol. 9:310–318. 2008. View Article : Google Scholar : PubMed/NCBI
|
16
|
Charles N, Watford WT, Ramos HL, et al:
Lyn kinase controls basophil GATA-3 transcription factor expression
and induction of Th2 cell differentiation. Immunity. 30:533–543.
2009. View Article : Google Scholar : PubMed/NCBI
|
17
|
Oh K, Shen T, Le Gros G and Min B:
Induction of Th2 type immunity in a mouse system reveals a novel
immunoregulatory role of basophils. Blood. 109:2921–2927.
2007.PubMed/NCBI
|
18
|
Denzel A, Maus UA, Gomez Rodriguez M, et
al: Basophils enhance immunological memory responses. Nat Immunol.
9:733–742. 2008. View
Article : Google Scholar : PubMed/NCBI
|
19
|
Gauchat JF, Henchoz S, Mazzei G, et al:
Induction of human IgE synthesis in B cells by mast cells and
basophils. Nature. 365:340–343. 1993. View
Article : Google Scholar : PubMed/NCBI
|
20
|
Gao E and Xiang J: Basophils for the
diagnosis of rheumatoid arthritis activity. Foreign Medical
Sciences (Clinical Biochemistry and Laboratory Medicine Volume).
3:401986.[(In Chinese)].
|
21
|
Chen Q, Tang P, Lan Q, Xie T, Wu P and Pan
Q: The number of activation of periphery basophils from patients
with rheumatoid arthritis. The 19th National Annual Conference of
the Chinese Rheumatology Association. 1622014.[(In Chinese)].
|
22
|
Askenase PW: Cutaneous basophil
hypersensitivity in contact-sensitized guinea pigs. I. Transfer
with immune serum. J Exp Med. 138:1144–1155. 1973. View Article : Google Scholar : PubMed/NCBI
|
23
|
Askenase PW, Graziano F and Worms M:
Basophils and mast cells. Immunobiology of cutaneous basophil
reactions. Monogr Allergy. 14:222–235. 1979.PubMed/NCBI
|
24
|
Ying S, Robinson DS, Meng Q, Barata LT,
McEuen AR, Buckley MG, et al: C-C chemokines in allergen-induced
late-phase cutaneous responses in atopic subjects: association of
eotaxin with early 6-hour eosinophils, and of eotaxin-2 and
monocyte chemoattractant protein-4 with the later 24-hour tissue
eosinophilia and relationship to basophils and other C-C chemokines
(monocyte chemoattractant protein-3 and RANTES). J Immunol.
163:3976–3984. 1999.PubMed/NCBI
|
25
|
Mitchell EB and Askenase PW: Basophils in
human disease. Clin Rev Allergy. 1:427–448. 1983.PubMed/NCBI
|
26
|
Obata K, Mukai K, Tsujimura Y, Ishiwata K,
Kawano Y, Minegishi Y, et al: Basophils are essential initiators of
a novel type of chronic allergic inflammation. Blood. 110:913–920.
2007. View Article : Google Scholar : PubMed/NCBI
|
27
|
Sokol CL, Barton GM, Farr AG and Medzhitov
R: A mechanism for the initiation of allergen-induced T helper type
2 responses. Nat Immunol. 9:310–318. 2008. View Article : Google Scholar : PubMed/NCBI
|
28
|
Charles N, Hardwick D, Daugas E, Illei GG
and Rivera J: Basophils and the T helper 2 environment can promote
the development of lupus nephritis. Nat Med. 16:701–707. 2010.
View Article : Google Scholar : PubMed/NCBI
|
29
|
Yamada H, Hirai K, Miyamasu M, Iikura M,
Misaki Y, Shoji S, Takaishi T, Kasahara T, Morita Y and Ito K:
Eotaxin is a potent chemotaxin for human basophils. Biochem Biophys
Res Commun. 231:365–368. 1997. View Article : Google Scholar : PubMed/NCBI
|
30
|
Uguccioni M, Mackay CR, Ochensberger B,
Loetscher P, Rhis S, LaRosa GJ, Rao P, Ponath PD, Baggiolini M and
Dahinden CA: High expression of the chemokine receptor CCR3 in
human blood basophils. Role in activation by eotaxin, MCP-4 and
other chemokines. J Clin Invest. 100:1137–1143. 1997. View Article : Google Scholar : PubMed/NCBI
|
31
|
Krieger M, Brunner T, Bischoff SC, von
Tscharner V, Walz A, Moser B, Baggiolini M and Dahinden CA:
Activation of human basophils through the IL-8 receptor. J Immunol.
149:2662–2667. 1992.PubMed/NCBI
|
32
|
Ochensberger B, Tassera L, Bifrare D, Rihs
S and Dahinden CA: Regulation of cytokine expression and
leukotriene formation in human basophils by growth factors,
chemokines and chemotactic agonists. Eur J Immunol. 29:11–22. 1999.
View Article : Google Scholar : PubMed/NCBI
|
33
|
Athreya BH, Moser G and Raghavan TE:
Increased circulating basophils in juvenile rheumatoid arthritis. A
preliminary report. Am J Dis Child. 129:935–937. 1975. View Article : Google Scholar : PubMed/NCBI
|
34
|
Athreya BH and Nichols WW: Basophils in
juvenile rheumatoid arthritis. Pediatr Res. 8:397. 1974. View Article : Google Scholar
|
35
|
Petty RE, Southwood TR, Baum J, et al:
Revision of the proposed classification criteria for juvenile
idiopathic arthritis: Durban 1997. J Rheumatol. 25:1991–1994.
1998.PubMed/NCBI
|
36
|
Petty RE, Southwood TR, Manners P, et al:
International League of Associations for Rheumatology
Classification of juvenile idiopathic arthritis: second revision,
Edmonton2001. J Rheumatol. 31:390–392. 2004.PubMed/NCBI
|
37
|
Seder RA, Paul WE, Dvorak AM, Sharkis SJ,
Kagey-Sobotka A, Niv Y, et al: Mouse splenic and bone marrow cell
populations that express high-affinity Fc epsilon receptors and
produce interleukin 4 are highly enriched in basophils. Proc Natl
Acad Sci USA. 88:2835–2839. 1991. View Article : Google Scholar : PubMed/NCBI
|
38
|
Schroeder JT, MacGlashan DW Jr and
Lichtenstein LM: Human basophils: mediator release and cytokine
production. Adv Immunol. 77:93–122. 2001. View Article : Google Scholar : PubMed/NCBI
|
39
|
Voehringer D, Shinkai K and Locksley RM:
Type 2 immunity reflects orchestrated recruitment of cells
committed to IL-4 production. Immunity. 20:267–277. 2004.
View Article : Google Scholar : PubMed/NCBI
|
40
|
Min B, Prout M, Hu-Li J, et al: Basophils
produce IL-4 and accumulate in tissues after infection with a
Th2-inducing parasite. J Exp Med. 200:507–517. 2004. View Article : Google Scholar : PubMed/NCBI
|
41
|
Bühring HJ, Seiffert M, Giesert C, et al:
The basophil activation marker defined by antibody 97A6 is
identical to the ectonucleotide pyrophosphatase/phosphodiesterase
3. Blood. 97:3303–3305. 2001. View Article : Google Scholar : PubMed/NCBI
|
42
|
Hauswirth AW, Natter S, Ghannadan M, et
al: Recombinant allergens promote expression of CD203c on basophils
in sensitized individuals. J Allergy Clin Immunol. 110:102–109.
2002. View Article : Google Scholar : PubMed/NCBI
|
43
|
Platz IJ, Binder M, Marxer A, Lischka G,
Valent P and Bühring HJ: Hymenoptera-venom-induced upregulation of
the basophil activation marker ecto-nucleotide
pyrophosphatase/phosphodiesterase 3 in sensitized individuals. Int
Arch Allergy Immunol. 126:335–342. 2001. View Article : Google Scholar : PubMed/NCBI
|
44
|
Bühring HJ, Streble A and Valent P: The
basophil-specific ectoenzyme E-NPP3 (CD203c) as a marker for cell
activation and allergy diagnosis. Int Arch Allergy Immunol.
133:317–329. 2004. View Article : Google Scholar : PubMed/NCBI
|
45
|
Hennersdorf F, Florian S, Jakob A, et al:
Identification of CD13, CD107a and CD164 as novel
basophil-activation markers and dissection of two response patterns
in time kinetics of IgE-dependent upregulation. Cell Res.
15:325–335. 2005. View Article : Google Scholar : PubMed/NCBI
|
46
|
Sonneck K, Baumgartner C, Rebuzzi L, et
al: Recombinant allergens promote expression of aminopeptidase-n
(CD13) on basophils in allergic patients. Int J Immunopathol
Pharmacol. 21:11–21. 2008.PubMed/NCBI
|
47
|
Chirumbolo S, Vella A, Ortolani R, et al:
Differential response of human basophil activation markers: a
multi-parameter flow cytometry approach. Clin Mol Allergy.
6:122008. View Article : Google Scholar : PubMed/NCBI
|
48
|
Sainte-Laudy J and Ouk C: Use of lipid
rafting for the analysis of human basophil activation by flow
cytometry. Inflamm Res. 59 (Suppl 2):S193–S195. 2010. View Article : Google Scholar : PubMed/NCBI
|
49
|
Valent P: Basophil activation antigens:
molecular mechanisms and clinical implications. Open Allergy J.
3:52–59. 2010. View Article : Google Scholar
|
50
|
Millauer N, Zuercher AW, Miescher SM, et
al: High IgE in rheumatoid arthritis (RA) patients is complexed
with anti-IgE autoantibodies. Clin Exp Immunol. 115:183–188. 1999.
View Article : Google Scholar : PubMed/NCBI
|
51
|
De Clerck LS, Struyf NJ, Bridts CH and
Stevens WJ: Activation of inflammatory cells by immune complexes
containing IgE in serum and synovial fluid of patients with
rheumatoid arthritis: a study using flow cytometric analysis. Ann
Rheum Dis. 50:379–382. 1991. View Article : Google Scholar : PubMed/NCBI
|
52
|
Van Offel JF, De Clerck LS and Kersschot
IE: Cholesterol crystals and IgE-containing immune complexes in
rheumatoid pericarditis. Clin Rheumatol. 10:78–80. 1991. View Article : Google Scholar : PubMed/NCBI
|
53
|
De Clerck LS, Struyf NJ, Bridts CH, et al:
IgE-containing immune complexes in synovial fluid of patients with
rheumatoid arthritis. Clin Rheumatol. 9:176–181. 1990. View Article : Google Scholar : PubMed/NCBI
|
54
|
Bartholomew JS, Evanson JM and Woolley DE:
Serum IgE anti-cartilage collagen antibodies in rheumatoid
patients. Rheumatol Int. 11:37–40. 1991. View Article : Google Scholar : PubMed/NCBI
|
55
|
Permin H, Wiik A and Djurup R:
Phagocytosis by normal polymorphonuclear leukocytes of immune
complexes from serum of patients with Felty's syndrome and
rheumatoid arthritis with special reference to IgE immune
complexes. Acta Pathol Microbiol Immunol Scand C. 92:37–42.
1984.PubMed/NCBI
|
56
|
Schuerwegh AJ, Ioan-Facsinay A, Dorjée AL,
et al: Evidence for a functional role of IgE anticitrullinated
protein antibodies in rheumatoid arthritis. Proc Natl Acad Sci USA.
107:2586–2591. 2010. View Article : Google Scholar : PubMed/NCBI
|
57
|
Müller B, Gimsa U, Mitchison NA, Radbruch
A, Sieper J and Yin Z: Modulating the Th1/Th2 balance in
inflammatory arthritis. Springer Semin Immunopathol. 20:181–196.
1998. View Article : Google Scholar : PubMed/NCBI
|
58
|
Nicholson LB and Kuchroo VK: Manipulation
of the Th1/Th2 balance in autoimmune disease. Curr Opin Immunol.
8:837–842. 1996. View Article : Google Scholar : PubMed/NCBI
|
59
|
Joosten LA, Lubberts E, Durez P, Helsen
MM, Jacobs MJ, Goldman M and van den Berg WB: Role of interleukin-4
and interleukin-10 in murine collagen-induced arthritis. Protective
effect of interleukin-4 and interleukin-10 treatment on cartilage
destruction. Arthritis Rheum. 40:249–260. 1997. View Article : Google Scholar : PubMed/NCBI
|