1
|
McMurray JJ, Adamopoulos S, Anker SD,
Auricchio A, Böhm M, Dickstein K, Falk V, Filippatos G, Fonseca C,
Gomez-Sanchez MA, et al: Task Force for the Diagnosis and Treatment
of Acute and Chronic Heart Failure 2012 of the European Society of
Cardiology; ESC Committee for Practice Guidelines: ESC guidelines
for the diagnosis and treatment of acute and chronic heart failure
2012: The Task Force for the Diagnosis and Treatment of Acute and
Chronic Heart Failure 2012 of the European Society of Cardiology.
Developed in collaboration with the Heart Failure Association (HFA)
of the ESC. Eur J Heart Fail. 14:803–869. 2012. View Article : Google Scholar : PubMed/NCBI
|
2
|
Gao S, Long CL, Wang RH and Wang H: K(ATP)
activation prevents progression of cardiac hypertrophy to failure
induced by pressure overload via protecting endothelial function.
Cardiovasc Res. 83:444–456. 2009. View Article : Google Scholar : PubMed/NCBI
|
3
|
Khazaei M, Moien-Afshari F and Laher I:
Vascular endothelial function in health and diseases.
Pathophysiology. 15:49–67. 2008. View Article : Google Scholar : PubMed/NCBI
|
4
|
Otsuka F, Finn AV, Yazdani SK, Nakano M,
Kolodgie FD and Virmani R: The importance of the endothelium in
atherothrombosis and coronary stenting. Nat Rev Cardiol. 9:439–453.
2012. View Article : Google Scholar : PubMed/NCBI
|
5
|
Pan X, Wang J, Pu Y, Yao J and Wang H:
Effect of puerarin on expression of ICAM-1 and TNF-alpha in kidneys
of diabetic rats. Med Sci Monit. 21:2134–2140. 2015. View Article : Google Scholar : PubMed/NCBI
|
6
|
Scalera F, Schlembach D and Beinder E:
Production of vasoactive substances by human umbilical vein
endothelial cells after incubation with serum from preeclamptic
patients. Eur J Obstet Gynecol Reprod Biol. 99:172–178. 2001.
View Article : Google Scholar : PubMed/NCBI
|
7
|
Tsikouris JP, Simoni J, Suarez JA,
Sutthiwan P, Ziska M and Meyerrose GE: Comparison of effects of
quinapril versus enalapril on vasoactive substances following acute
myocardial infarction. Am J Cardiol. 94:641–643. 2004. View Article : Google Scholar : PubMed/NCBI
|
8
|
Vanhoutte PM, Shimokawa H, Feletou M and
Tang EH: Endothelial dysfunction and vascular disease - a
thirthieth anniversary update. Acta Physiol (Oxf). 26:123–124.
2015.
|
9
|
Butta NV, Fernández-Bello I, López-Longo
FJ and Jiménez-Yuste V: Endothelial dysfunction and altered
coagulation as mediators of thromboembolism in Behçet disease.
Semin Thromb Hemost. 41:621–628. 2015. View Article : Google Scholar : PubMed/NCBI
|
10
|
Mudau M, Genis A, Lochner A and Strijdom
H: Endothelial dysfunction: the early predictor of atherosclerosis.
Cardiovasc J Afr. 23:222–231. 2012. View Article : Google Scholar : PubMed/NCBI
|
11
|
Kadowaki D, Anraku M, Sakaya M, Hirata S,
Maruyama T and Otagiri M: Olmesartan protects endothelial cells
against oxidative stress-mediated cellular injury. Clin Exp
Nephrol. 19:1007–1014. 2015. View Article : Google Scholar : PubMed/NCBI
|
12
|
Zingman LV, Alekseev AE, Hodgson-Zingman
DM and Terzic A: ATP-sensitive potassium channels: metabolic
sensing and cardioprotection. J Appl Physiol 1985. 103:1888–1893.
2007. View Article : Google Scholar : PubMed/NCBI
|
13
|
Chen X, Han W, Zhang Y, Cui W, Pan Z, Jin
X, Long C and Wang H: The molecular pathway of ATP-sensitive
potassium channel in endothelial cells for mediating arteriole
relaxation. Life Sci. 137:164–169. 2015. View Article : Google Scholar : PubMed/NCBI
|
14
|
Seino S and Miki T: Physiological and
pathophysiological roles of ATP-sensitive K+ channels.
Prog Biophys Mol Biol. 81:133–176. 2003. View Article : Google Scholar : PubMed/NCBI
|
15
|
Watanuki M, Horie M, Tsuchiya K, Obayashi
K and Sasayama S: Endothelin-1 inhibition of cardiac ATP-sensitive
K+ channels via pertussis-toxin-sensitive G-proteins.
Cardiovasc Res. 33:123–130. 1997. View Article : Google Scholar : PubMed/NCBI
|
16
|
Vita JA and Keaney JF Jr: Endothelial
function: a barometer for cardiovascular risk? Circulation.
106:640–642. 2002. View Article : Google Scholar : PubMed/NCBI
|
17
|
Marti CN, Gheorghiade M, Kalogeropoulos
AP, Georgiopoulou VV, Quyyumi AA and Butler J: Endothelial
dysfunction, arterial stiffness, and heart failure. J Am Coll
Cardiol. 60:1455–1469. 2012. View Article : Google Scholar : PubMed/NCBI
|
18
|
Jandeleit-Dahm KA and Watson AM: The
endothelin system and endothelin receptor antagonists. Curr Opin
Nephrol Hypertens. 21:66–71. 2012. View Article : Google Scholar : PubMed/NCBI
|
19
|
Sun Y, Ye L, Jiang C, Jiang J, Hong H and
Qiu L: Over-expression of HSPA12B protects mice against myocardium
ischemic/reperfusion injury through a PPARγ-dependent PI3K/Akt/eNOS
pathway. Am J Transl Res. 7:2724–2737. 2015.PubMed/NCBI
|
20
|
Liu YH, Carretero OA, Cingolani OH, Liao
TD, Sun Y, Xu J, Li LY, Pagano PJ, Yang JJ and Yang XP: Role of
inducible nitric oxide synthase in cardiac function and remodeling
in mice with heart failure due to myocardial infarction. Am J
Physiol Heart Circ Physiol. 289:H2616–H2623. 2005. View Article : Google Scholar : PubMed/NCBI
|
21
|
Scherrer-Crosbie M, Ullrich R, Bloch KD,
Nakajima H, Nasseri B, Aretz HT, Lindsey ML, Vançon AC, Huang PL,
Lee RT, et al: Endothelial nitric oxide synthase limits left
ventricular remodeling after myocardial infarction in mice.
Circulation. 104:1286–1291. 2001. View Article : Google Scholar : PubMed/NCBI
|
22
|
Starling RC: Inducible nitric oxide
synthase in severe human heart failure: impact of mechanical
unloading. J Am Coll Cardiol. 45:1425–1427. 2005. View Article : Google Scholar : PubMed/NCBI
|
23
|
Jones SP, Greer JJ, van Haperen R, Duncker
DJ, de Crom R and Lefer DJ: Endothelial nitric oxide synthase
overexpression attenuates congestive heart failure in mice. Proc
Natl Acad Sci USA. 100:4891–4896. 2003. View Article : Google Scholar : PubMed/NCBI
|