1
|
Morin DP, Bernard ML, Madias C, Rogers PA,
Thihalolipavan S and Estes NA III: The state of the art: Atrial
fibrillation epidemiology, prevention, and treatment. Mayo Clin
Proc. 91:1778–1810. 2016. View Article : Google Scholar : PubMed/NCBI
|
2
|
Violi F and Loffredo L: Thromboembolism or
atherothromboembolism in atrial fibrillation? Circ Arrhythm
Electrophysiol. 5:1053–1055. 2012. View Article : Google Scholar : PubMed/NCBI
|
3
|
Odutayo A, Wong CX, Hsiao AJ, Hopewell S,
Altman DG and Emdin CA: Atrial fibrillation and risks of
cardiovascular disease, renal disease, and death: Systematic review
and meta-analysis. BMJ. 354:i44822016. View Article : Google Scholar : PubMed/NCBI
|
4
|
Lubitz SA, Yin X, Fontes JD, Magnani JW,
Rienstra M, Pai M, Villalon ML, Vasan RS, Pencina MJ, Levy D, et
al: Association between familial atrial fibrillation and risk of
new-onset atrial fibrillation. JAMA. 304:2263–2269. 2010.
View Article : Google Scholar : PubMed/NCBI
|
5
|
Amano M, Nakayama M and Kaibuchi K:
Rho-kinase/ROCK: A key regulator of the cytoskeleton and cell
polarity. Cytoskeleton (Hoboken). 67:545–554. 2010. View Article : Google Scholar : PubMed/NCBI
|
6
|
Wei L, Taffet GE, Khoury DS, Bo J, Li Y,
Yatani A, Delaughter MC, Klevitsky R, Hewett TE, Robbins J, et al:
Disruption of Rho signaling results in progressive atrioventricular
conduction defects while ventricular function remains preserved.
FASEB J. 18:857–859. 2004. View Article : Google Scholar : PubMed/NCBI
|
7
|
Kajikawa M, Noma K, Maruhashi T, Mikami S,
Iwamoto Y, Iwamoto A, Matsumoto T, Hidaka T, Kihara Y, Chayama K,
et al: Rho-associated kinase activity is a predictor of
cardiovascular outcomes. Hypertension. 63:856–864. 2014. View Article : Google Scholar : PubMed/NCBI
|
8
|
Demiryürek S, Kara AF, Celik A, Babül A,
Tarakcioglu M and Demiryürek AT: Effects of fasudil, a Rho-kinase
inhibitor, on myocardial preconditioning in anesthetized rats. Eur
J Pharmacol. 527:129–1240. 2005. View Article : Google Scholar : PubMed/NCBI
|
9
|
Demiryürek S, Kara AF, Celik A,
Tarakçioğlu M, Bagci C and Demiryürek AT: Effects of Y-27632, a
selective Rho-kinase inhibitor, on myocardial preconditioning in
anesthetized rats. Biochem Pharmacol. 69:49–58. 2005. View Article : Google Scholar : PubMed/NCBI
|
10
|
Hata T, Soga J, Hidaka T, Idei N, Fujii Y,
Fujimura N, Mikami S, Maruhashi T, Kihara Y, Chayama K, et al:
Calcium channel blocker and Rho-associated kinase activity in
patients with hypertension. J Hypertens. 29:373–379. 2011.
View Article : Google Scholar : PubMed/NCBI
|
11
|
Ocaranza MP, Gabrielli L, Mora I, Garcia
L, McNab P, Godoy I, Braun S, Córdova S, Castro P, Novoa U, et al:
Markedly increased Rho-kinase activity in circulating leukocytes in
patients with chronic heart failure. Am Heart J. 161:931–937. 2011.
View Article : Google Scholar : PubMed/NCBI
|
12
|
Liu PY, Chen JH, Lin LJ and Liao JK:
Increased Rho kinase activity in a Taiwanese population with
metabolic syndrome. J Am Coll Cardiol. 49:1619–1624. 2007.
View Article : Google Scholar : PubMed/NCBI
|
13
|
Nohria A, Grunert ME, Rikitake Y, Noma K,
Prsic A, Ganz P, Liao JK and Creager MA: Rho kinase inhibition
improves endothelial function in human subjects with coronary
artery disease. Circ Res. 99:1426–1432. 2006. View Article : Google Scholar : PubMed/NCBI
|
14
|
Maruhashi T, Noma K, Fujimura N, Kajikawa
M, Matsumoto T, Hidaka T, Nakashima A, Kihara Y, Liao JK and
Higashi Y: Exogenous nitric oxide inhibits Rho-associated kinase
activity in patients with angina pectoris: A randomized controlled
trial. Hypertens Res. 38:485–490. 2015. View Article : Google Scholar : PubMed/NCBI
|
15
|
Li X, Wu X, Li H, Chen H, Wang Y, Li W,
Ding X and Hong X: Increased Rho kinase activity predicts worse
cardiovascular outcome in ST-segment elevation myocardial
infarction patients. Cardiol J. 23:456–464. 2016. View Article : Google Scholar : PubMed/NCBI
|
16
|
Chen HC, Chang JP, Chang TH, Lin YS, Huang
YK, Pan KL, Fang CY, Chen CJ, Ho WC and Chen MC: Enhanced
expression of ROCK in left atrial myocytes of mitral regurgitation:
A potential mechanism of myolysis. BMC Cardiovasc Disord.
15:332015. View Article : Google Scholar : PubMed/NCBI
|
17
|
Wennerberg K, Forget MA, Ellerbroek SM,
Arthur WT, Burridge K, Settleman J, Der CJ and Hansen SH: Rnd
proteins function as RhoA antagonists by activating p190 RhoGAP.
Curr Biol. 13:1106–1115. 2003. View Article : Google Scholar : PubMed/NCBI
|
18
|
Riento K, Villalonga P, Garg R and Ridley
A: Function and regulation of RhoE. Biochem Soc Trans. 33:649–651.
2005. View Article : Google Scholar : PubMed/NCBI
|
19
|
Jie W, Andrade KC, Lin X, Yang X, Yue X
and Chang J: Pathophysiological functions of Rnd3/RhoE. Compr
Physiol. 6:169–186. 2016.
|
20
|
Yang X, Wang T, Lin X, Yue X, Wang Q, Wang
G, Fu Q, Ai X, Chiang DY, Miyake CY, et al: Genetic deletion of
Rnd3/RhoE results in mouse heart calcium leakage through
upregulation of protein kinase A signaling. Circ Res. 116:e1–e10.
2015. View Article : Google Scholar : PubMed/NCBI
|
21
|
Tsai FC, Chang GJ, Hsu YJ, Lin YM, Lee YS,
Chen WJ, Kuo CT and Yeh YH: Proinflammatory gene expression in
patients undergoing mitral valve surgery and maze ablation for
atrial fibrillation. J Thorac Cardiovasc Surg. 151:1673–1682 e5.
2016. View Article : Google Scholar : PubMed/NCBI
|
22
|
Wherlock M and Mellor H: The RhoGTPase
family: A Racs to Wrchs story. J Cell Sci. 115:239–240.
2002.PubMed/NCBI
|
23
|
Ji W and Rivero F: Atypical Rho GTPases of
the RhoBTB subfamily: Roles in vesicle trafficking and
tumorigenesis. Cells. 5:E282016. View Article : Google Scholar : PubMed/NCBI
|
24
|
Pai SY, Kim C and Williams DA: RacGTPases
in human diseases. Dis Markers. 29:177–187. 2010. View Article : Google Scholar : PubMed/NCBI
|
25
|
Adam O, Frost G, Custodis F, Sussman MA,
Schäfers HJ, Böhm M and Laufs U: Role of Rac1 GTPase activation in
atrial fibrillation. J Am Coll Cardiol. 50:359–367. 2007.
View Article : Google Scholar : PubMed/NCBI
|
26
|
Loirand G, Sauzeau V and Pacaud P: Small G
proteins in the cardiovascular system: Physiological and
pathological aspects. Physiol Rev. 93:1659–1720. 2013. View Article : Google Scholar : PubMed/NCBI
|
27
|
Shimano M, Shibata R, Inden Y, Yoshida N,
Uchikawa T, Tsuji Y and Murohara T: Reactive oxidative metabolites
are associated with atrial conduction disturbance in patients with
atrial fibrillation. Heart Rhythm. 6:935–940. 2009. View Article : Google Scholar : PubMed/NCBI
|
28
|
Kim YH, Lim DS, Lee JH, Shim WJ, Ro YM,
Park GH, Becker KG, Cho-Chung YS and Kim MK: Gene expression
profiling of oxidative stress on atrial fibrillation in humans. Exp
Mol Med. 35:336–349. 2003. View Article : Google Scholar : PubMed/NCBI
|
29
|
Jin L, Ying Z and Webb RC: Activation of
Rho/Rho kinase signaling pathway by reactive oxygen species in rat
aorta. Am J Physiol Heart Circ Physiol. 287:H1495–H1500. 2004.
View Article : Google Scholar : PubMed/NCBI
|
30
|
Higashi M, Shimokawa H, Hattori T, Hiroki
J, Mukai Y, Morikawa K, Ichiki T, Takahashi S and Takeshita A:
Long-term inhibition of Rho-kinase suppresses angiotensin
II-induced cardiovascular hypertrophy in rats in vivo: Effect on
endothelial NAD(P)H oxidase system. Circ Res. 93:767–775. 2003.
View Article : Google Scholar : PubMed/NCBI
|
31
|
Lin H, Yin X, Lunetta KL, Dupuis J,
McManus DD, Lubitz SA, Magnani JW, Joehanes R, Munson PJ, Larson
MG, et al: Whole blood gene expression and atrial fibrillation: The
framingham heart study. PLoS One. 9:e967942014. View Article : Google Scholar : PubMed/NCBI
|
32
|
Raman K, Aeschbacher S, Bossard M,
Hochgruber T, Zimmermann AJ, Kaufmann BA, Pumpol K, Rickenbacker P,
Paré G and Conen D: Whole blood gene expression differentiates
between atrial fibrillation and sinus rhythm after cardioversion.
PLoS One. 11:e01575502016. View Article : Google Scholar : PubMed/NCBI
|
33
|
Gurses KM, Kocyigit D, Yalcin MU, Canpinar
H, Yorgun H, Sahiner ML, Kaya EB, Oto MA, Ozer N, Guc D and Aytemir
K: Monocyte Toll-like receptor expression in patients with atrial
fibrillation. Am J Cardiol. 117:1463–1467. 2016. View Article : Google Scholar : PubMed/NCBI
|
34
|
Düzen IV, Yavuz F, Vuruskan E, Saracoglu
E, Poyraz F, Göksülük H, Candemir B and Demiryürek S: Leukocyte TRP
channel gene expressions in patients with non-valvular atrial
fibrillation. Sci Rep. 7:92722017. View Article : Google Scholar : PubMed/NCBI
|