1
|
Lai TS, Wang CY, Pan SC, Huang TM, Lin MC,
Lai CF, Wu CH, Wu VC and Chien KL: National Taiwan University
Hospital Study Group on Acute Renal Failure (NSARF). Risk of
developing severe sepsis after acute kidney injury: A
population-based cohort study. Crit Care. 17(R231)2013.PubMed/NCBI View
Article : Google Scholar
|
2
|
Weng L, Zeng XY, Yin P, Wang LJ, Wang CY,
Jiang W, Zhou MG and Du B: China Critical Care Clinical Trials
Group (CCCCTG). Sepsis-related mortality in China: A descriptive
analysis. Intensive Care Med. 44:1071–1080. 2018.PubMed/NCBI View Article : Google Scholar
|
3
|
Poston JT and Koyner JL: Sepsis associated
acute kidney injury. BMJ. 364(k4891)2019.PubMed/NCBI View Article : Google Scholar
|
4
|
Messaris E, Memos N, Chatzigianni E,
Kataki A, Nikolopoulou M, Manouras A, Albanopoulos K,
Konstadoulakis MM and Bramis J: Apoptotic death of renal tubular
cells in experimental sepsis. Surg Infect (Larchmt). 9:377–388.
2008.PubMed/NCBI View Article : Google Scholar
|
5
|
Priante G, Gianesello L, Ceol M, Del Prete
D and Anglani F: Cell death in the kidney. Int J Mol Sci.
20(3598)2019.PubMed/NCBI View Article : Google Scholar
|
6
|
Ye Z, Zhang L, Li R, Dong W, Liu S, Li Z,
Liang H, Wang L, Shi W, Malik AB, et al: Caspase-11 mediates
pyroptosis of tubular epithelial cells and septic acute kidney
injury. Kidney Blood Press Res. 44:465–478. 2019.PubMed/NCBI View Article : Google Scholar
|
7
|
Bergsbaken T, Fink SL and Cookson BT:
Pyroptosis: Host cell death and inflammation. Nat Rev Microbiol.
7:99–109. 2009.PubMed/NCBI View Article : Google Scholar
|
8
|
Man SM, Karki R and Kanneganti TD:
Molecular mechanisms and functions of pyroptosis, inflammatory
caspases and inflammasomes in infectious diseases. Immunol Rev.
277:61–75. 2017.PubMed/NCBI View Article : Google Scholar
|
9
|
Jorgensen I, Rayamajhi M and Miao EA:
Programmed cell death as a defence against infection. Nat Rev
Immunol. 17:151–164. 2017.PubMed/NCBI View Article : Google Scholar
|
10
|
Sedlackova L and Korolchuk VI:
Mitochondrial quality control as a key determinant of cell
survival. Biochim Biophys Acta Mol Cell Res. 1866:575–587.
2019.PubMed/NCBI View Article : Google Scholar
|
11
|
Li HB, Zhang XZ, Sun Y, Zhou Q, Song JN,
Hu ZF, Li Y, Wu JN, Guo Y, Zhang Y, et al: HO-1/PINK1 regulated
mitochondrial fusion/fission to inhibit pyroptosis and attenuate
septic acute kidney injury. Biomed Res Int.
2020(2148706)2020.PubMed/NCBI View Article : Google Scholar
|
12
|
Pfanner N, Warscheid B and Wiedemann N:
Mitochondrial protein organization: From biogenesis to networks and
function. Nat Rev Mol Cell Biol. 20:267–284. 2019.PubMed/NCBI View Article : Google Scholar
|
13
|
Supinski GS, Schroder EA and Callahan LA:
Mitochondria and critical illness. Chest. 157:310–322.
2020.PubMed/NCBI View Article : Google Scholar
|
14
|
Bolisetty S, Zarjou A and Agarwal A: Heme
oxygenase 1 as a therapeutic target in acute kidney injury. Am J
Kidney Dis. 69:531–545. 2017.PubMed/NCBI View Article : Google Scholar
|
15
|
Suliman HB, Keenan JE and Piantadosi CA:
Mitochondrial quality-control dysregulation in conditional
HO-1-/- mice. JCI Insight. 2(e89676)2017.PubMed/NCBI View Article : Google Scholar
|
16
|
Chen X, Wang Y, Xie X, Chen H, Zhu Q, Ge
Z, Wei H, Deng J, Xia Z and Lian Q: Heme oxygenase-1 reduces
sepsis-induced endoplasmic reticulum stress and acute lung injury.
Mediators Inflamm. 2018(9413876)2018.PubMed/NCBI View Article : Google Scholar
|
17
|
Tang C, Han H, Yan M, Zhu S, Liu J, Liu Z,
He L, Tan J, Liu Y, Liu H, et al: PINK1-PRKN/PARK2 pathway of
mitophagy is activated to protect against renal
ischemia-reperfusion injury. Autophagy. 14:880–897. 2018.PubMed/NCBI View Article : Google Scholar
|
18
|
Ding W, Yousefi K and Shehadeh LA:
Isolation, characterization, and high throughput extracellular flux
analysis of mouse primary renal tubular epithelial cells. J Vis
Exp. (57718)2018.PubMed/NCBI View
Article : Google Scholar
|
19
|
Livak KJ and Schmittgen TD: Analysis of
relative gene expression data using real-time quantitative PCR and
the 2(-Delta Delta C(T)) method. Methods. 25:402–408.
2001.PubMed/NCBI View Article : Google Scholar
|
20
|
Mattila PM, Nietosvaara YA, Ustinov JK,
Renkonen RL and Häyry PJ: Antigen expression in different
parenchymal cell types of rat kidney and heart. Kidney Int.
36:228–233. 1989.PubMed/NCBI View Article : Google Scholar
|
21
|
Van Kooten C, Lam S and Daha MR:
Isolation, culture, characterization and use of human renal tubular
epithelial cells. J Nephrol. 14:204–210. 2001.PubMed/NCBI
|
22
|
Shankar-Hari M, Phillips GS, Levy ML,
Seymour CW, Liu VX, Deutschman CS, Angus DC and Rubenfeld GD:
Developing a new definition and assessing new clinical criteria for
septic shock: For the third international consensus definitions for
sepsis and septic shock (sepsis-3). JAMA. 315:775–787.
2016.PubMed/NCBI View Article : Google Scholar
|
23
|
Cecconi M, Evans L, Levy M and Rhodes A:
Sepsis and septic shock. Lancet. 392:75–87. 2018.PubMed/NCBI View Article : Google Scholar
|
24
|
Plotnikov EY, Brezgunova AA, Pevzner IB,
Zorova LD, Manskikh VN, Popkov VA, Silachev DN and Zorov DB:
Mechanisms of LPS-induced acute kidney injury in neonatal and adult
rats. Antioxidants (Basel). 7(105)2018.PubMed/NCBI View Article : Google Scholar
|
25
|
Quoilin C, Mouithys-Mickalad A, Duranteau
J, Gallez B and Hoebeke M: Endotoxin-induced basal respiration
alterations of renal HK-2 cells: A sign of pathologic metabolism
down-regulation. Biochem Biophys Res Commun. 423:350–354.
2012.PubMed/NCBI View Article : Google Scholar
|
26
|
Tang YQ and Li L: Development strategy and
application of animal model of sepsis. Chin J Exp Surg.
12:1433–1434. 2006.(In Chinese).
|
27
|
Peerapornratana S, Manrique-Caballero CL,
Gómez H and Kellum JA: Acute kidney injury from sepsis: Current
concepts, epidemiology, pathophysiology, prevention and treatment.
Kidney Int. 96:1083–1099. 2019.PubMed/NCBI View Article : Google Scholar
|
28
|
Chen F, Lu J, Yang X, Xiao B, Chen H, Pei
W, Jin Y, Wang M, Li Y, Zhang J, et al: Acetylbritannilactone
attenuates contrast-induced acute kidney injury through its
anti-pyroptosis effects. Biosci Rep. 40(BSR20193253)2020.PubMed/NCBI View Article : Google Scholar
|
29
|
Shi J, Zhao Y, Wang Y, Gao W, Ding J, Li
P, Hu L and Shao F: Inflammatory caspases are innate immune
receptors for intracellular LPS. Nature. 514:187–192.
2014.PubMed/NCBI View Article : Google Scholar
|
30
|
Broz P: Immunology: Caspase target drives
pyroptosis. Nature. 526:642–643. 2015.PubMed/NCBI View Article : Google Scholar
|
31
|
Friedman JR and Nunnari J: Mitochondrial
form and function. Nature. 505:335–343. 2014.PubMed/NCBI View Article : Google Scholar
|
32
|
Yu W, Sheng M, Xu R, Yu J, Cui K, Tong J,
Shi L, Ren H and Du H: Berberine protects human renal proximal
tubular cells from hypoxia/reoxygenation injury via inhibiting
endoplasmic reticulum and mitochondrial stress pathways. J Transl
Med. 11(24)2013.PubMed/NCBI View Article : Google Scholar
|
33
|
Murphy MP and Hartley RC: Mitochondria as
a therapeutic target for common pathologies. Nat Rev Drug Discov.
17:865–886. 2018.PubMed/NCBI View Article : Google Scholar
|
34
|
Yu JB, Zhou F, Yao SL, Tang ZH, Wang M and
Chen HR: Effect of heme oxygenase-1 on the kidney during septic
shock in rats. Transl Res. 153:283–287. 2009.PubMed/NCBI View Article : Google Scholar
|
35
|
Yin H, Li X, Yuan B, Zhang B, Hu S, Gu H,
Jin X and Zhu J: Heme oxygenase-1 ameliorates LPS-induced acute
lung injury correlated with downregulation of interleukin-33. Int
Immunopharmacol. 11:2112–2117. 2011.PubMed/NCBI View Article : Google Scholar
|
36
|
Abraham NG, Lin JH, Schwartzman ML, Levere
RD and Shibahara S: The physiological significance of heme
oxygenase. Int J Biochem. 20:543–558. 1988.PubMed/NCBI View Article : Google Scholar
|
37
|
Gozzelino R, Jeney V and Soares MP:
Mechanisms of cell protection by heme oxygenase-1. Annu Rev
Pharmacol Toxicol. 50:323–354. 2010.PubMed/NCBI View Article : Google Scholar
|
38
|
Kozakowska M, Dulak J and Józkowicz A:
Heme oxygenase-1-more than the cytoprotection. Postepy Biochem.
61:147–158. 2015.PubMed/NCBI(In Polish).
|
39
|
Cai ZY, Sheng ZX and Yao H: Pachymic acid
ameliorates sepsis-induced acute kidney injury by suppressing
inflammation and activating the Nrf2/HO-1 pathway in rats. Eur Rev
Med Pharmacol Sci. 21:1924–1931. 2017.PubMed/NCBI
|
40
|
Bayne AN and Trempe J: Mechanisms of
PINK1, ubiquitin and parkin interactions in mitochondrial quality
control and beyond. Cell Mol Life Sci. 76:4589–4611.
2019.PubMed/NCBI View Article : Google Scholar
|
41
|
Lin Q, Li S, Jiang N, Shao X, Zhang M, Jin
H, Zhang Z, Shen J, Zhou Y, Zhou W, et al: PINK1-parkin pathway of
mitophagy protects against contrast-induced acute kidney injury via
decreasing mitochondrial ROS and NLRP3 inflammasome activation.
Redox Biol. 26(101254)2019.PubMed/NCBI View Article : Google Scholar
|
42
|
Leites EP and Morais VA: Mitochondrial
quality control pathways: PINK1 acts as a gatekeeper. Biochem
Biophys Res Commun. 500:45–50. 2018.PubMed/NCBI View Article : Google Scholar
|
43
|
Yu JB, Shi J, Zhang Y, Gong LR, Dong SA,
Cao XS, Wu LL and Wu LN: Electroacupuncture ameliorates acute renal
injury in lipopolysaccharide-stimulated rabbits via induction of
HO-1 through the PI3K/Akt/Nrf2 pathways. PLoS One.
10(e0141622)2015.PubMed/NCBI View Article : Google Scholar
|