1
|
El-Serag HB and Mason AC: Rising incidence
of hepatocellular carcinoma in the United States. N Engl J Med.
340:745–750. 1999. View Article : Google Scholar : PubMed/NCBI
|
2
|
Parkin DM, Bray F, Ferlay J and Pisani P:
Estimating the world cancer burden: Globocan 2000. Int J Cancer.
94:153–156. 2001. View
Article : Google Scholar : PubMed/NCBI
|
3
|
lkai I, Yamaoka Y, Yamamoto Y, Ozaki N,
Sakai Y, Satoh S, Shinkura N and Yamamoto M: Surgical intervention
for patients with stage IV-A hepatocellular carcinoma without lymph
node metastasis: proposal as a standard therapy. Ann Surg.
227:433–439. 1998. View Article : Google Scholar
|
4
|
Lee JS, Chu IS, Heo J, Calvisi DF, Sun Z,
Roskams T, Durnez A, Demetris AJ and Thorgeirsson SS:
Classification and prediction of survival in hepatocellular
carcinoma by gene expression profiling. Hepatology. 40:667–676.
2004. View Article : Google Scholar : PubMed/NCBI
|
5
|
Song HY, Liu YK, Feng JT, Cui JF, Dai Z,
Zhang LJ, Feng JX, Shen HL and Tang ZY: Proteomic analysis on
metastasis-associated proteins of human hepatocellular carcinoma
tissues. J Cancer Res Clin Oncol. 132:92–98. 2006. View Article : Google Scholar
|
6
|
Yang CS and Wang ZY: Tea and cancer. J
Natl Cancer Inst. 85:1038–1049. 1993. View Article : Google Scholar : PubMed/NCBI
|
7
|
Balentine DA, Wiseman SA and Bouwens LC:
The chemistry of tea flavonoids. Crit Rev Food Sci Nutr.
37:693–704. 1997. View Article : Google Scholar
|
8
|
Mukhtar H and Ahmad N: Green tea in
chemoprevention of cancer. Toxicol Sci. 52(Suppl 2): 111–117. 1999.
View Article : Google Scholar
|
9
|
Singh BN, Shankar S and Srivastava RK:
Green tea catechin, epigallocatechin-3-gallate (EGCG): mechanisms,
perspectives and clinical applications. Biochem Pharmacol.
82:1807–1821. 2011. View Article : Google Scholar : PubMed/NCBI
|
10
|
Thawonsuwan J, Kiron V, Satoh S, Panigrahi
A and Verlhac V: Epigallocatechin-3-gallate (EGCG) affects the
antioxidant and immune defense of the rainbow trout, Oncorhynchus
mykiss. Fish Physiol Biochem. 36:687–697. 2010. View Article : Google Scholar
|
11
|
Mukhtar H and Ahmad N: Tea polyphenols:
prevention of cancer and optimizing health. Am J Clin Nutr.
71(Suppl 6): 1698S–1702S. 2000.PubMed/NCBI
|
12
|
Thangapazham RL, Singh AK, Sharma A,
Warren J, Gaddipati J and Maheshwari RK: Green tea polyphenols and
its constituent epigallocatechin gallate inhibits proliferation of
human breast cancer cells in vitro and in vivo. Cancer Lett.
245:232–241. 2007. View Article : Google Scholar
|
13
|
Stuart EC, Scandlyn MJ and Rosengren RJ:
Role of epigallocatechin gallate (EGCG) in the treatment of breast
and prostate cancer. Life Sci. 79:2329–2336. 2006. View Article : Google Scholar : PubMed/NCBI
|
14
|
Ahmad N, Feyes DK, Nieminen AL, Agarwal R
and Mukhtar H: Green tea constituent epigallocatechin-3-gallate and
induction of apoptosis and cell cycle arrest in human carcinoma
cells. J Natl Cancer Inst. 89:1881–1886. 1997. View Article : Google Scholar : PubMed/NCBI
|
15
|
Masuda M, Suzui M and Weinstein IB:
Effects of epigallocatechin-3-gallate on growth, epidermal growth
factor receptor signaling pathways, gene expression, and
chemosensitivity in human head and neck squamous cell carcinoma
cell lines. Clin Cancer Res. 7:4220–4229. 2001.PubMed/NCBI
|
16
|
Nihal M, Ahmad N, Mukhtar H and Wood GS:
Anti-proliferative and proapoptotic effects of
(−)-epigallocatechin-3-gallate on human melanoma: possible
implications for the chemoprevention of melanoma. Int J Cancer.
114:513–521. 2005. View Article : Google Scholar
|
17
|
Gupta S, Hussain T and Mukhtar H:
Molecular pathway for (−)-epigallocatechin-3-gallate-induced cell
cycle arrest and apoptosis of human prostate carcinoma cells. Arch
Biochem Biophys. 410:177–185. 2003. View Article : Google Scholar : PubMed/NCBI
|
18
|
Wei DZ, Yang JY, Liu JW and Tong WY:
Inhibition of liver cancer cell proliferation and migration by a
combination of (−)-epigallocatechin-3-gallate and ascorbic acid. J
Chemother. 15:591–595. 2003. View Article : Google Scholar
|
19
|
Uesato S, Kitagawa Y, Kamishimoto M,
Kumagai A, Hori H and Nagasawa H: Inhibition of green tea catechins
against the growth of cancerous human colon and hepatic epithelial
cells. Cancer Lett. 170:41–44. 2001. View Article : Google Scholar : PubMed/NCBI
|
20
|
Nishikawa T, Nakajima T, Moriguchi M, Jo
M, Sekoguchi S, Ishii M, et al: A green tea polyphenol,
epigalocatechin-3-gallate, induces apoptosis of human
hepatocellular carcinoma, possibly through inhibition of Bcl-2
family proteins. J Hepatol. 44:1074–1082. 2006. View Article : Google Scholar : PubMed/NCBI
|
21
|
Wyllie AH, Kerr JF and Currie AR: Cell
death: the significance of apoptosis. Int Rev Cytol. 68:251–306.
1980. View Article : Google Scholar : PubMed/NCBI
|
22
|
Soldatenkov VA and Smulson M: Poly
(ADP-ribose) polymerase in DNA damage-response pathway:
implications for radiation oncology. Int J Cancer. 90:59–67. 2000.
View Article : Google Scholar : PubMed/NCBI
|
23
|
Wei MC, Zong WX, Cheng EH, Lindsten T,
Panoutsakopoulou V, Ross AJ, et al: Proapoptotic BAX and BAK: a
requisite gateway to mitochondrial dysfunction and death. Science.
292:727–730. 2001. View Article : Google Scholar : PubMed/NCBI
|
24
|
Green DR and Reed JC: Mitochondria and
apoptosis. Science. 281:1309–1312. 1998. View Article : Google Scholar : PubMed/NCBI
|
25
|
Marsden VS, O’Connor L, O’Reilly LA, Silke
J, Metcalf D, Ekert PG, et al: Apoptosis initiated by
Bcl-2-regulated caspase activation independently of the cytochrome
c/Apaf-1/caspase-9 apoptosome. Nature. 419:634–637. 2002.
View Article : Google Scholar : PubMed/NCBI
|
26
|
Tsujimoto Y: Cell death regulation by the
Bcl-2 protein family in the mitochondria. J Cell Physiol.
195:158–167. 2003. View Article : Google Scholar : PubMed/NCBI
|
27
|
Kowaltowski AJ, Vercesi AE and Fiskum G:
Bcl-2 prevents mitochondrial permeability transition and cytochrome
c release via maintenance of reduced pyridine nucleotides. Cell
Death Differ. 7:903–910. 2000. View Article : Google Scholar
|
28
|
Reed JC: Proapoptotic multidomain
Bcl-2/Bax-family proteins: mechanisms, physiological roles, and
therapeutic opportunities. Cell Death Differ. 13:1378–1386. 2006.
View Article : Google Scholar : PubMed/NCBI
|
29
|
Schuler M, Bossy-Wetzel E, Goldstein JC,
Fitzgerald P and Green DR: p53 induces apoptosis by caspase
activation through mitochondrial cytochrome c release. J Biol Chem.
275:7337–7342. 2000. View Article : Google Scholar : PubMed/NCBI
|
30
|
Miyashita T and Reed JC: Tumor suppressor
p53 is a direct transcriptional activator of the human bax gene.
Cell. 80:293–299. 1995. View Article : Google Scholar : PubMed/NCBI
|
31
|
Ahmad N, Cheng P and Mukhtar H: Cell cycle
dysregulation by green tea polyphenol epigallocatechin-3-gallate.
Biochem Biophys Res Commun. 275:328–334. 2000. View Article : Google Scholar : PubMed/NCBI
|
32
|
Weinert TA and Hartwell LH: The RAD9 gene
controls the cell cycle response to DNA damage in Saccharomyces
cerevisiae. Science. 241:317–322. 1988. View Article : Google Scholar : PubMed/NCBI
|
33
|
Smerdon MJ, Kastan MB and Lieberman MW:
Distribution of repair-incorporated nucleotides and nucleosome
rearrangement in the chromatin of normal and xeroderma pigmentosum
human fibroblasts. Biochemistry. 18:3732–3739. 1979. View Article : Google Scholar : PubMed/NCBI
|
34
|
Kastan MB, Canman CE and Christopher J:
p53, cell cycle control and apoptosis: implications for cancer.
Cancer Metastasis Rev. 14:3–15. 1995. View Article : Google Scholar : PubMed/NCBI
|
35
|
Chow HH, Cai Y, Hakim IA, Crowell JA,
Shahi F, Brooks CA, Dorr RT, Hara Y and Alberts DS:
Pharmacokinetics and safety of green tea polyphenols after
multiple-dose administration of epigallocatechin gallate and
polyphenon E in healthy individuals. Clin Cancer Res. 9:3312–3319.
2003.PubMed/NCBI
|
36
|
Chow HH, Cai Y, Alberts DS, et al: Phase I
pharmacokinetic study of tea polyphenols following single-dose
administration of epigallocatechin gallate and polyphenon E. Cancer
Epidemiol Biomarkers Prev. 10:53–58. 2001.PubMed/NCBI
|
37
|
Li QS, Wang CY, Han GZ, Zou LL, Li L and
Li N: Application of LC-MS/MS method in pharmacokinetic study of
multi-components from tea polyphenols in rats. Chin J New Drugs.
20:817–823. 2011.
|