1
|
Laky D, Constantinescu S, Filipescu G,
Ratea E and Zeană C: Morphophysiological studies in experimental
myocardial aggressions with isoproterenol. Note I. Morphological
aspects. Morphol Embryol (Bucur). 29:273–277. 1983.PubMed/NCBI
|
2
|
Kung HF and Blau M: Subcutaneous
isoproterenol: A convenient rat model for early detection of
myocardial necrosis. J Nucl Med. 19:948–951. 1978.PubMed/NCBI
|
3
|
Parikh NI, Gona P, Larson MG, Fox CS,
Benjamin EJ, Murabito JM, O'Donnell CJ, Vasan RS and Levy D:
Long-term trends in myocardial infarction incidence and case
fatality in the National Heart, Lung, and Blood Institute's
Framingham Heart study. Circulation. 119:1203–1210. 2009.
View Article : Google Scholar : PubMed/NCBI
|
4
|
Sutton MG and Sharpe N: Left ventricular
remodeling after myocardial infarction: Pathophysiology and
therapy. Circulation. 101:2981–2988. 2000. View Article : Google Scholar : PubMed/NCBI
|
5
|
Gajarsa JJ and Kloner RA: Left ventricular
remodeling in the post-infarction heart: A review of cellular,
molecular mechanisms, and therapeutic modalities. Heart Fail Rev.
16:13–21. 2011. View Article : Google Scholar : PubMed/NCBI
|
6
|
Yoon HJ, Jeong MH, Jeong Y, Kim KH, Song
JE, Cho JY, Jang SY, Jeong HC, Lee KH, Park KH, et al: Progressive
dilation of the left atrium and ventricle after acute myocardial
infarction is associated with high mortality. Korean Circ J.
43:731–738. 2013. View Article : Google Scholar : PubMed/NCBI
|
7
|
Olivetti G, Abbi R, Quaini F, Kajstura J,
Cheng W, Nitahara JA, Quaini E, Di Loreto C, Beltrami CA, Krajewski
S, et al: Apoptosis in the failing human heart. N Engl J Med.
336:1131–1141. 1997. View Article : Google Scholar : PubMed/NCBI
|
8
|
Heusch G, Libby P, Gersh B, Yellon D, Böhm
M, Lopaschuk G and Opie L: Cardiovascular remodelling in coronary
artery disease and heart failure. Lancet. 383:1933–1943. 2014.
View Article : Google Scholar : PubMed/NCBI
|
9
|
Kuznetsova T, Herbots L, Jin Y,
Stolarz-Skrzypek K and Staessen JA: Systolic and diastolic left
ventricular dysfunction: From risk factors to overt heart failure.
Expert Rev Cardiovasc Ther. 8:251–258. 2010. View Article : Google Scholar : PubMed/NCBI
|
10
|
Song ZH, Dai SJ, Li HQ, Bi KS and Feng D:
Study on the compatibility of composite herbal medicines of the
lingguizhugan decoction. Zhongguo Zhong Yao Za Zhi. 27:760–762.
2002.(In Chinese). PubMed/NCBI
|
11
|
Council NR: Guide for the Care and Use of
Laboratory Animals. The National Academies Press; Washington, DC:
1996
|
12
|
Remme WJ: Pharmacological modulation of
cardiovascular remodeling: A guide to heart failure therapy.
Cardiovasc Drugs Ther. 17:349–360. 2003. View Article : Google Scholar : PubMed/NCBI
|
13
|
Swynghedauw B: Molecular mechanisms of
myocardial remodeling. Physiol Rev. 79:215–262. 1999.PubMed/NCBI
|
14
|
Mitsi AC, Hatzistergos KE, Niokou D, Pappa
L, Baltogiannis GG, Tsalikakis DG, Papalois A, Kyriakides ZS,
Malamou-Mitsi V and Kolettis TM: Early, intracoronary growth
hormone administration attenuates ventricular remodeling in a
porcine model of myocardial infarction. Growth Horm IGF Res.
16:93–100. 2006. View Article : Google Scholar : PubMed/NCBI
|
15
|
Maisch B: Ventricular remodeling.
Cardiology. 87 Suppl 1:S2–S10. 1996. View Article : Google Scholar
|
16
|
Rona G, Chappel CI, Balazs T and Gaudry R:
An infarct-like myocardial lesion and other toxic manifestations
produced by isoproterenol in the rat. AMA Arch Pathol. 67:443–455.
1959.PubMed/NCBI
|
17
|
Tisné-Versailles J, Constantin M, Lamar JC
and Pourrias B: Cardiotoxicity of high doses of isoproterenol on
cardiac haemodynamics and metabolism in SHR and WKY rats. Arch Int
Pharmacodyn Ther. 273:142–154. 1985.PubMed/NCBI
|
18
|
Ji K, Minakawa M, Fukui K, Suzuki Y and
Fukuda I: Increased superoxide radical with a decrease in vascular
endothelial growth factor and inducible nitric oxide synthase level
leads to the progression of left ventricular hypertrophy in a
pressure-overload rat heart model. Ann Thorac Cardiovasc Surg.
14:210–217. 2008.PubMed/NCBI
|
19
|
Hohimer AR, Davis LE and Hatton DC:
Repeated daily injections and osmotic pump infusion of
isoproterenol cause similar increases in cardiac mass but have
different effects on blood pressure. Can J Physiol Pharmacol.
83:191–197. 2005. View
Article : Google Scholar : PubMed/NCBI
|
20
|
Krenning G, Zeisberg EM and Kalluri R: The
origin of fibroblasts and mechanism of cardiac fibrosis. J Cell
Physiol. 225:631–637. 2010. View Article : Google Scholar : PubMed/NCBI
|
21
|
Kong P, Christia P and Frangogiannis NG:
The pathogenesis of cardiac fibrosis. Cell Mol Life Sci.
71:549–574. 2014. View Article : Google Scholar : PubMed/NCBI
|
22
|
Wynn TA and Ramalingam TR: Mechanisms of
fibrosis: Therapeutic translation for fibrotic disease. Nat Med.
18:1028–1040. 2012. View
Article : Google Scholar : PubMed/NCBI
|
23
|
Krzesinski JM, Rorive G and Van
Cauwenberge H: Hypertension and left ventricular hypertrophy. Acta
Cardiol. 51:143–154. 1996.PubMed/NCBI
|
24
|
Weber KT and Brilla CG: Pathological
hypertrophy and cardiac interstitium. Fibrosis and
renin-angiotensin-aldosterone system. Circulation. 83:1849–1865.
1991. View Article : Google Scholar : PubMed/NCBI
|
25
|
Teerlink JR, Pfeffer JM and Pfeffer MA:
Progressive ventricular remodeling in response to diffuse
isoproterenol-induced myocardial necrosis in rats. Circ Res.
75:105–113. 1994. View Article : Google Scholar : PubMed/NCBI
|
26
|
Wang HJ, Wang W, Cornish KG, Rozanski GJ
and Zucker IH: Cardiac sympathetic afferent denervation attenuates
cardiac remodeling and improves cardiovascular dysfunction in rats
with heart failure. Hypertension. 64:745–755. 2014. View Article : Google Scholar : PubMed/NCBI
|
27
|
Semenov AG, Seferian KR, Tamm NN,
Artem'eva MM, Postnikov AB, Bereznikova AV, Kara AN, Medvedeva NA
and Katrukha AG: Human pro-B-type natriuretic peptide is processed
in the circulation in a rat model. Clin Chem. 57:883–890. 2011.
View Article : Google Scholar : PubMed/NCBI
|
28
|
Dogan H, Sarikaya S, Neijmann ST, Uysal E,
Yucel N, Ozucelik DN, Okuturlar Y, Solak S, Sever N and Ayan C:
N-terminal pro-B-type natriuretic peptide as a marker of blunt
cardiac contusion in trauma. Int J Clin Exp Pathol. 8:6786–6792.
2015.PubMed/NCBI
|
29
|
Tziakas DN, Chalikias GK, Hatzinikolaou
EI, Stakos DA, Tentes IK, Kortsaris A, Hatseras DI and Kaski JC:
N-terminal pro-B-type natriuretic peptide and matrix
metalloproteinases in early and late left ventricular remodeling
after acute myocardial infarction. Am J Cardiol. 96:31–34. 2005.
View Article : Google Scholar : PubMed/NCBI
|
30
|
Parthasarathy A, Gopi V, Devi K, M S,
Balaji N and Vellaichamy E: Aminoguanidine inhibits ventricular
fibrosis and remodeling process in isoproterenol-induced
hypertrophied rat hearts by suppressing ROS and MMPs. Life Sci.
118:15–26. 2014. View Article : Google Scholar : PubMed/NCBI
|
31
|
Pryor WA and Stanley JP: Letter: A
suggested mechanism for the production of malonaldehyde during the
autoxidation of polyunsaturated fatty acids. Nonenzymatic
production of prostaglandin endoperoxides during autoxidation. J
Org Chem. 40:3615–3617. 1975. View Article : Google Scholar : PubMed/NCBI
|
32
|
Rathore N, John S, Kale M and Bhatnagar D:
Lipid peroxidation and antioxidant enzymes in isoproterenol induced
oxidative stress in rat tissues. Pharmacol Res. 38:297–303. 1998.
View Article : Google Scholar : PubMed/NCBI
|
33
|
Del Rio D, Stewart AJ and Pellegrini N: A
review of recent studies on malondialdehyde as toxic molecule and
biological marker of oxidative stress. Nutr Metab Cardiovasc Dis.
15:316–328. 2005. View Article : Google Scholar : PubMed/NCBI
|
34
|
Sayer G and Bhat G: The
renin-angiotensin-aldosterone system and heart failure. Cardiol
Clin. 32:21–32, vii. 2014. View Article : Google Scholar : PubMed/NCBI
|
35
|
Gregori M, Tocci G, Marra A, Pignatelli G,
Santolamazza C, Befani A, Ciavarella GM, Ferrucci A and Paneni F:
Inadequate RAAS suppression is associated with excessive left
ventricular mass and systo-diastolic dysfunction. Clin Res Cardiol.
102:725–733. 2013. View Article : Google Scholar : PubMed/NCBI
|
36
|
Neves MF, Amiri F, Virdis A, Diep QN and
Schiffrin EL; CIHR Multidisciplinary Research Group on
Hypertension, : Role of aldosterone in angiotensin II-induced
cardiac and aortic inflammation, fibrosis, and hypertrophy. Can J
Physiol Pharmacol. 83:999–1006. 2005. View
Article : Google Scholar : PubMed/NCBI
|