1
|
Stylianopoulos T: The solid mechanics of
cancer and strategies for improved therapy. J Biomech Eng.
139:2017. View Article : Google Scholar : PubMed/NCBI
|
2
|
Fukumura D and Jain RK: Tumor
microenvironment abnormalities: Causes, consequences, and
strategies to normalize. J Cell Biochem. 101:937–949. 2007.
View Article : Google Scholar : PubMed/NCBI
|
3
|
Goel S, Duda DG, Xu L, Munn LL, Boucher Y,
Fukumura D and Jain RK: Normalization of the vasculature for
treatment of cancer and other diseases. Physiol Rev. 91:1071–1121.
2011. View Article : Google Scholar : PubMed/NCBI
|
4
|
Martin JD, Fukumura D, Duda DG, Boucher Y
and Jain RK: Reengineering the tumor microenvironment to alleviate
hypoxia and overcome cancer heterogeneity. Cold Spring Harb
Perspect Med. 6:2016. View Article : Google Scholar
|
5
|
Jain RK: Normalizing tumor
microenvironment to treat cancer: Bench to bedside to biomarkers. J
Clin Oncol. 31:2205–2218. 2013. View Article : Google Scholar : PubMed/NCBI
|
6
|
Vassilakopoulou M, Psyrri A and Argiris A:
Targeting angiogenesis in head and neck cancer. Oral Oncol.
51:409–415. 2015. View Article : Google Scholar : PubMed/NCBI
|
7
|
Tas SW, Maracle CX, Balogh E and Szekanecz
Z: Targeting of proangiogenic signalling pathways in chronic
inflammation. Nat Rev Rheumatol. 12:111–122. 2016. View Article : Google Scholar : PubMed/NCBI
|
8
|
Simon T, Gagliano T and Giamas G: Direct
effects of anti-angiogenic therapies on tumor cells: VEGF
signaling. Trends Mol Med. 23:282–292. 2017. View Article : Google Scholar : PubMed/NCBI
|
9
|
Ye W: The complexity of translating
anti-angiogenesis therapy from basic science to the clinic. Dev
Cell. 37:114–125. 2016. View Article : Google Scholar : PubMed/NCBI
|
10
|
Al-Bataineh O, Jenne J and Huber P:
Clinical and future applications of high intensity focused
ultrasound in cancer. Cancer Treat Rev. 38:346–353. 2012.
View Article : Google Scholar : PubMed/NCBI
|
11
|
Kennedy JE: High-intensity focused
ultrasound in the treatment of solid tumours. Nat Rev Cancer.
5:321–327. 2005. View
Article : Google Scholar : PubMed/NCBI
|
12
|
Brown MR, Farquhar-Smith P, Williams JE,
ter Haar G and deSouza NM: The use of high-intensity focused
ultrasound as a novel treatment for painful conditions-a
description and narrative review of the literature. Br J Anaesth.
115:520–530. 2015. View Article : Google Scholar : PubMed/NCBI
|
13
|
Pan H, Zhou W and Wang S: Pulsed focused
ultrasound stimulates the release of tumor biomarkers into the
blood circulation. Radiology. 285:1058–1060. 2017. View Article : Google Scholar : PubMed/NCBI
|
14
|
Hariharan P, Myers MR and Banerjee RK:
HIFU procedures at moderate intensities-effect of large blood
vessels. Phys Med Biol. 52:3493–3513. 2007. View Article : Google Scholar : PubMed/NCBI
|
15
|
Qiao Y, Yin H, Li Z and Wan M: Cavitation
distribution within large phantom vessel and mechanical damage
formed on surrounding vessel wall. Ultrason Sonochem. 20:1376–1383.
2013. View Article : Google Scholar : PubMed/NCBI
|
16
|
Mizrahi N, Zhou EH, Lenormand G, Krishnan
R, Weihs D, Butler JP, Weitz DA, Fredberg JJ and Kimmel E: Low
intensity ultrasound perturbs cytoskeleton dynamics. Soft matter.
8:2438–2443. 2012. View Article : Google Scholar : PubMed/NCBI
|
17
|
Rutten S, Nolte PA, Korstjens CM and
Klein-Nulend J: Low-intensity pulsed ultrasound affects RUNX2
immunopositive osteogenic cells in delayed clinical fracture
healing. Bone. 45:862–869. 2009. View Article : Google Scholar : PubMed/NCBI
|
18
|
Hitchcock KE and Holland CK:
Ultrasound-assisted thrombolysis for stroke therapy: Better
thrombus break-up with bubbles. Stroke. 41:S50–S53. 2010.
View Article : Google Scholar : PubMed/NCBI
|
19
|
Zhang Z, Chen J, Chen L, Yang X, Zhong H,
Qi X, Bi Y and Xu K: Low frequency and intensity ultrasound induces
apoptosis of brain glioma in rats mediated by caspase-3, Bcl-2, and
survivin. Brain Res. 1473:25–34. 2012. View Article : Google Scholar : PubMed/NCBI
|
20
|
Zhou XY, Wu SY, Zhang ZC, Wang F, Yang YL,
Li M and Wei XZ: Low-intensity pulsed ultrasound promotes
endothelial cell-mediated osteogenesis in a conditioned medium
coculture system with osteoblasts. Medicine (Baltimore).
96:e83972017. View Article : Google Scholar : PubMed/NCBI
|
21
|
Marquez RT and Xu L: Bcl-2: Beclin 1
complex: Multiple, mechanisms regulating autophagy/apoptosis toggle
switch. Am J Cancer Res. 2:214–221. 2012.PubMed/NCBI
|
22
|
Rovetta F, Stacchiotti A, Consiglio A,
Cadei M, Grigolato PG, Lavazza A, Rezzani R and Aleo MF: ER
signaling regulation drives the switch between autophagy and
apoptosis in NRK-52E cells exposed to cisplatin. Exp Cell Res.
318:238–250. 2012. View Article : Google Scholar : PubMed/NCBI
|
23
|
Furusawa Y, Zhao QL, Hassan MA, Tabuchi Y,
Takasaki I, Wada S and Kondo T: Ultrasound-induced apoptosis in the
presence of Sonazoid and associated alterations in gene expression
levels: A possible therapeutic application. Cancer Lett.
288:107–115. 2010. View Article : Google Scholar : PubMed/NCBI
|
24
|
Gao Q, Walmsley AD, Cooper PR and Scheven
BA: Ultrasound stimulation of different dental stem cell
populations: Role of mitogen-activated protein kinase signaling. J
Endod. 42:425–431. 2016. View Article : Google Scholar : PubMed/NCBI
|
25
|
Lee GS, Park JH, Shin US and Kim HW:
Direct deposited porous scaffolds of calcium phosphate cement with
alginate for drug delivery and bone tissue engineering. Acta
Biomater. 7:3178–3186. 2011. View Article : Google Scholar : PubMed/NCBI
|
26
|
Mason TJ: Therapeutic ultrasound an
overview. Ultrason Sonochem. 18:847–852. 2011. View Article : Google Scholar : PubMed/NCBI
|
27
|
Hou R, Xu Y, Lu Q, Zhang Y and Hu B:
Effect of low-frequency low-intensity ultrasound with microbubbles
on prostate cancer hypoxia. Tumour Biol. 39:10104283177192752017.
View Article : Google Scholar : PubMed/NCBI
|
28
|
Chen H, Brayman AA, Bailey MR and Matula
TJ: Blood vessel rupture by cavitation. Urol Res. 38:321–326. 2010.
View Article : Google Scholar : PubMed/NCBI
|
29
|
Elmore S: Apoptosis: A review of
programmed cell death. Toxicol Pathol. 35:495–516. 2007. View Article : Google Scholar : PubMed/NCBI
|
30
|
Chen Y and Brandizzi F: IRE1: ER stress
sensor and cell fate executor. Trends Cell Biol. 23:547–555. 2013.
View Article : Google Scholar : PubMed/NCBI
|
31
|
Bhutia SK, Dash R, Das SK, Azab B, Su ZZ,
Lee SG, Grant S, Yacoub A, Dent P, Curiel DT, et al: Mechanism of
autophagy to apoptosis switch triggered in prostate cancer cells by
antitumor cytokine melanoma differentiation-associated gene
7/interleukin-24. Cancer Res. 70:3667–3676. 2010. View Article : Google Scholar : PubMed/NCBI
|
32
|
Wu H, Che X, Zheng Q, Wu A, Pan K, Shao A,
Wu Q, Zhang J and Hong Y: Caspases: A molecular switch node in the
crosstalk between autophagy and apoptosis. Int J Biol Sci.
10:1072–1083. 2014. View Article : Google Scholar : PubMed/NCBI
|
33
|
Obata T, Brown GE and Yaffe MB: MAP kinase
pathways activated by stress: The p38 MAPK pathway. Crit Care Med.
28:N67–N77. 2000. View Article : Google Scholar : PubMed/NCBI
|
34
|
Whitmarsh AJ: A central role for p38 MAPK
in the early transcriptional response to stress. BMC Biol.
8:472010. View Article : Google Scholar : PubMed/NCBI
|
35
|
Trempolec N, Dave-Coll N and Nebreda AR:
SnapShot: p38 MAPK substrates. Cell. 152:924–924.e1. 2013.
View Article : Google Scholar : PubMed/NCBI
|
36
|
Grossi V, Peserico A, Tezil T and Simone
C: p38alpha MAPK pathway: A key factor in colorectal cancer therapy
and chemoresistance. World J Gastroenterol. 20:9744–9758. 2014.
View Article : Google Scholar : PubMed/NCBI
|
37
|
Olson JM and Hallahan AR: p38 MAP kinase:
A convergence point in cancer therapy. Trends Mol Med. 10:125–129.
2004. View Article : Google Scholar : PubMed/NCBI
|
38
|
Pang H, Cai L, Yang Y, Chen X, Sui G and
Zhao C: Knockdown of osteopontin chemosensitizes MDA-MB-231 cells
to cyclophosphamide by enhancing apoptosis through activating p38
MAPK pathway. Cancer Biother Radiopharm. 26:165–173. 2011.
View Article : Google Scholar : PubMed/NCBI
|
39
|
Sui X, Kong N, Ye L, Han W, Zhou J, Zhang
Q, He C and Pan H: p38 and JNK MAPK pathways control the balance of
apoptosis and autophagy in response to chemotherapeutic agents.
Cancer Lett. 344:174–179. 2014. View Article : Google Scholar : PubMed/NCBI
|
40
|
Matsui H, Fukuno N, Kanda Y, Kantoh Y,
Chida T, Nagaura Y, Suzuki O, Nishitoh H, Takeda K, Ichijo H, et
al: The expression of Fn14 via mechanical stress-activated JNK
contributes to apoptosis induction in osteoblasts. J Biol Chem.
289:6438–6450. 2014. View Article : Google Scholar : PubMed/NCBI
|
41
|
Mu C, Lv T, Wang Z, Ma S, Ma J, Liu J, Yu
J and Mu J: Mechanical stress stimulates the osteo/odontoblastic
differentiation of human stem cells from apical papilla via erk 1/2
and JNK MAPK pathways. Biomed Res Int. 2014:4943782014. View Article : Google Scholar : PubMed/NCBI
|
42
|
Xu T, Gu J, Li C, Guo X, Tu J, Zhang D,
Sun W and Kong X: Low-intensity pulsed ultrasound suppresses
proliferation and promotes apoptosis via p38 MAPK signaling in rat
visceral preadipocytes. Am J Transl Res. 10:948–956.
2018.PubMed/NCBI
|
43
|
Jiang Q, Li F, Shi K, Wu P, An J, Yang Y
and Xu C: Involvement of p38 in signal switching from autophagy to
apoptosis via the PERK/eIF2α/ATF-4 axis in selenite-treated NB4
cells. Cell Death Dis. 5:e12702014. View Article : Google Scholar : PubMed/NCBI
|
44
|
Rozpedek W, Pytel D, Mucha B, Leszczynska
H, Diehl JA and Majsterek I: The role of the PERK/eIF2α/ATF-4/CHOP
signaling pathway in tumor progression during endoplasmic reticulum
stress. Curr Mol Med. 16:533–544. 2016. View Article : Google Scholar : PubMed/NCBI
|