1
|
Globocan. 2008, http://wwwdep.iarc.fr/globocan/database.htm.
|
2
|
Burd EM: Human papillomavirus and cervical
cancer. Clin Microbiol Rev. 16:1–17. 2003. View Article : Google Scholar : PubMed/NCBI
|
3
|
Virmani AK, Muller C, Rathi A,
Zoechbauer-Mueller S, Mathis M and Gazdar AF: Aberrant methylation
during cervical carcinogenesis. Clinical Cancer Res. 7:584–589.
2001.PubMed/NCBI
|
4
|
Hirama T and Koeffer HP: Role of the
cyclin dependent kinase inhibitors in the development of cancer.
Blood. 86:841–854. 1995.PubMed/NCBI
|
5
|
Hannon GJ and Beach D: p15INK4B is a
potential effector of TGF-beta-induced cell cycle arrest. Nature.
371:257–261. 1994. View
Article : Google Scholar : PubMed/NCBI
|
6
|
Liggett WH Jr and Sidransky D: Role of the
p16 tumor suppressor gene in cancer. J Clin Oncol. 16:1197–1206.
1998.PubMed/NCBI
|
7
|
Merlo A, Herman JG, Mao L, Lee DJ,
Gabrielson E, Burger PC, Baylin SB and Sidransky D: 5′ CpG island
methylation is associated with transcriptional silencing of the
tumour suppressor p16/CDKN2/MTS1 in human cancers. Nat Med.
1:686–692. 1995.
|
8
|
Zhang SJ, Endo S, Ichikawa T, Washiyama K
and Kumanishi T: Frequent deletion and 5 CpG island methylation of
the p16 gene in primary malignant lymphoma of the brain. Cancer
Res. 58:1231–1237. 1998.PubMed/NCBI
|
9
|
Esteller M, Corn PG, Baylin SB and Herman
JG: A gene hypermethylation profile of human cancer. Cancer Res.
61:3225–3229. 2001.PubMed/NCBI
|
10
|
Rocco JW and Sidransky D:
p16(MTS-1/CDKN2/INK4a) in cancer progression. Exp Cell Res.
264:42–55. 2001. View Article : Google Scholar : PubMed/NCBI
|
11
|
Feng Q, Balasubramanian A, Hawes SE, Toure
P, Sow PS, Dem A, Dembele B, Critchlow CW, Xi L, Lu H, McIntosh MW,
Young AM and Kiviat NB: Detection of hypermethylated genes in women
with and without cervical neoplasia. J Natl Cancer Inst.
97:273–282. 2005. View Article : Google Scholar : PubMed/NCBI
|
12
|
D’Costa J, Saranath D, Dedhia P, Sanghvi V
and Mehta RA: Detection of HPV-16 genome in human oral cancers and
potentially malignant lesions from India. Oral Oncol. 34:413–420.
1998.PubMed/NCBI
|
13
|
Park JS, Dong SM, Kim HS, Lee JY, Jong Um
S, Park IS, Kim SJ and Namkoong SE: Detection of p16 gene
alteration in cervical cancer using tissue microdissection and LOH
study. Cancer Lett. 136:101–108. 1999. View Article : Google Scholar : PubMed/NCBI
|
14
|
Yeh KT, Chang GA, Lin TH, Wang YF, Tien N,
Chang JY, Chen CJ and Shih MC: Epigenetics changes of tumor
suppressor genes, p15, p16, VHIL and p53 in oral
cancer. Oncol Rep. 10:659–663. 2003.PubMed/NCBI
|
15
|
Esteller M, Tortola S, Toyota M, Capella
G, Peinado MA, Baylin SB and Herman JG: Hypermethylation-associated
inactivation of p14ARF is independent of
p16INK4a methylation and p53
mutational status. Cancer Res. 60:129–133. 2000.
|
16
|
Herman JG, Jen J, Merlo A and Baylin SB:
Hypermethylation associated inactivation indicates a tumor
suppressor role for p15INK4b. Cancer Res. 56:722–727.
1996.PubMed/NCBI
|
17
|
Perrone F, Tabano S, Colombo F, Dagrada G,
Birindelli S, Gronchi A, Colecchia M, Pierotti MA and Pilotti S:
p15INK4b, p14ARF
and p16INK4a inactivation in sporadic and
neurofibromatosis type 1-related malignant peripheral nerve sheath
tumors. Clinical Cancer Research. 9:4132–4138. 2003.
|
18
|
Walboomers JM, Jacobs MV, Manos MM, Bosch
FX, Kummer JA, Shah KV, Snijders PJ, Peto J, Meijer CJ and Muñoz N:
Human papillomavirus is a necessary cause of invasive cervical
cancer worldwide. J Pathol. 189:12–19. 1999. View Article : Google Scholar : PubMed/NCBI
|
19
|
Tay SK and Tay KJ: Passive cigarette
smoking is a risk factor for cervical neoplasia. Gynecol Oncol.
93:116–120. 2004. View Article : Google Scholar : PubMed/NCBI
|
20
|
Louie KS, de Sanjose S, Diaz M,
Castellsagué X, Herrero R, Meijer CJ, Shah K, Franceschi S, Muñoz N
and Bosch FX; International Agency for Research on Cancer
Multicenter Cervical Cancer Study Group. Early age at first sexual
intercourse and early pregnancy are risk factors for cervical
cancer in developing countries. Br J Cancer. 100:1191–1197. 2009.
View Article : Google Scholar : PubMed/NCBI
|
21
|
Jha AK, Nikbakht M, Parashar G,
Shrivastava A, Capalash N and Kaur J: Reversal of hypermethylation
and reactivation of the RARβ2 gene by natural compounds in cervical
cancer lines. Folia Biol (Praha). 56:195–200. 2010.PubMed/NCBI
|
22
|
Jha AK, Kumar S, Nikbakht M, Sharma V and
Kaur J: Epigenetics and its role in ageing and cancer. J Med Med
Sci. 2:696–713. 2011.
|
23
|
Narayan G, Arias-Pulido H, Koul S, Vargas
H, Zhang FF, Villella J, Schneider A, Terry MB, Mansukhani M and
Murty VV: Frequent promoter methylation of CDH1, DAPK, RARβ and
HIC1 genes in carcinoma of cervix uteri: its relationship to
clinical outcome. Mol Cancer. 2:242003.PubMed/NCBI
|
24
|
Ishida E, Nakamura M, Ikuta M, Shimada K,
Matsuyoshi S, Kirita T and Konishi N: Promotor hypermethylation of
p14ARF is a key alteration for progression of oral
squamous cell carcinoma. Oral Oncol. 41:614–622. 2005.
|
25
|
Kurakawa E, Shimamoto T and Utsumi K:
Hypermethylation of p16INK4a and
p15INK4b gene in non-small cell lung cancer. Int J Oncol.
19:277–281. 2001.
|
26
|
Jeong DH, Youm MY, Kim NY, Lee KB, Sung
MS, Yoon HK and Kim KT: Promoter methylation of p16, DAPK, CDH1 and
TIMP-3 genes in cervical cancer: correlation with clinicopathologic
characteristics. Int J Gynecol Cancer. 16:1234–1240. 2006.
View Article : Google Scholar : PubMed/NCBI
|
27
|
Tripathi A, Banerjee S, Roy A,
Roychowdhury S and Panda CK: Alterations of the p16 gene in uterine
cervical carcinoma from Indian patients. Int J Gynecol Cancer.
13:472–479. 2003. View Article : Google Scholar : PubMed/NCBI
|
28
|
Attaleb M, El hamadani W, Khyatti M,
Benbacer L, Benchekroun N, Benider A, Amrani M and El Mzibri M:
Status of p16INK4a and E-cadherin gene promoter
methylation in Moroccan patients with cervical carcinoma. Oncol
Res. 184:185–192. 2009.
|
29
|
Lea JS, Coleman R, Kurien A, Schorge JO,
Miller SD, Minna JD and Muller YC: Aberrant p16 methylation is a
biomarker for tobacco exposure in cervical squamous cell
carcinogenesis. Am J Obstet Gynecol. 190:674–679. 2004. View Article : Google Scholar : PubMed/NCBI
|