1
|
Ostrom QT, Gittleman H, Farah P, Ondracek
A, Chen Y, Wolinsky Y, Stroup NE, Kruchko C and Barnholtz-Sloan JS:
CBTRUS statistical report: Primary brain and central nervous system
tumors diagnosed in the United States in 2006–2010. Neuro-Oncology.
15(Suppl 2): ii1–ii56. 2013. View Article : Google Scholar : PubMed/NCBI
|
2
|
van Lohuizen M, Verbeek S, Scheljen B,
Wientjens E, van der Guidon H and Berns A: Identification of
cooperating oncogenes in E mu-myc transgenic mice by provirus
tagging. Cell. 65:737–752. 1991. View Article : Google Scholar : PubMed/NCBI
|
3
|
Datta S, Hoenerhoff MJ, Bommi P, Sainger
R, Guo WJ, Dimri M, Band H, Band V, Green JE and Dimri GP: Bmi-1
cooperates with H-Ras to transform human mammary epithelial cells
via dysregulation of multiple growth-regulatory pathways. Cancer
Res. 67:10286–10295. 2007. View Article : Google Scholar : PubMed/NCBI
|
4
|
Lukacs RU, Memarzadeh S, Wu H and Witte
ON: Bmi-1 is a crucial regulator of prostate stem cell self-renewal
and malignant transformation. Cell Stem Cell. 7:682–693. 2010.
View Article : Google Scholar : PubMed/NCBI
|
5
|
Wang X, Venugopal C, Manoranjan B,
McFarlane N, O'Farrell E, Nolte S, Gunnarsson T, Hollenberg R,
Kwiecien J, Northcott P, et al: Sonic hedgehog regulates Bmi1 in
human medulloblastoma brain tumor-initiating cells. Oncogene.
31:187–199. 2012. View Article : Google Scholar : PubMed/NCBI
|
6
|
Bachmann IM, Puntervoll HE, Otte AP and
Akslen LA: Loss of BMI-1 expression is associated with clinical
progress of malignant melanoma. Mod Pathol. 21:583–590. 2008.
View Article : Google Scholar : PubMed/NCBI
|
7
|
Cui H, Hu B, Li T, Ma J, Alam G, Gunning
WT and Ding HF: Bmi-1 is essential for the tumorigenicity of
neuroblastoma cells. Am J Pathol. 170:1370–1378. 2007. View Article : Google Scholar : PubMed/NCBI
|
8
|
Engelsen IB, Mannelqvist M, Stefansson IM,
Carter SL, Beroukhim R, Øyan AM, Otte AP, Kalland KH, Akslen LA and
Salvesen HB: Low BMI-1 expression is associated with an activated
BMI-1-driven signature, vascular invasion and hormone receptor loss
in endometrial carcinoma. Br J Cancer. 98:1662–1669. 2008.
View Article : Google Scholar : PubMed/NCBI
|
9
|
Vonlanthen S, Heighway J, Altermatt HJ,
Gugger M, Kappeler A, Borner MM, van Lohuizen M and Betticher DC:
The bmi-1 oncoprotein is differentially expressed in non-small cell
lung cancer and correlates with INK4A-ARF locus expression. Br J
Cancer. 84:13722001. View Article : Google Scholar : PubMed/NCBI
|
10
|
Vormittag L, Thurnher D, Geleff S, Pammer
J, Heiduschka G, Brunner M, Grasl MCH and Erovic BM: Co-expression
of Bmi-1 and podoplanin predicts overall survival in patients with
squamous cell carcinoma of the head and neck treated with radio
(chemo) therapy. Int J Radiat Oncol Biol Phys. 73:913–918. 2009.
View Article : Google Scholar : PubMed/NCBI
|
11
|
Abdouh M, Facchino S, Chatoo W, Balasingam
V, Ferreira J and Bernier G: BMI1 sustains human glioblastoma
multiforme stem cell renewal. J Neurosci. 29:8884–8896. 2009.
View Article : Google Scholar : PubMed/NCBI
|
12
|
Häyry V, Tynninen O, Haapasalo HK, Wölfer
J, Paulus W, Hasselblatt M, Sariola H, Paetau A, Sarna S, Niemelä
M, et al: Stem cell protein BMI-1 is an independent marker for poor
prognosis in oligodendroglial tumours. Neuropathol Appl Neurobiol.
34:555–563. 2008. View Article : Google Scholar : PubMed/NCBI
|
13
|
Häyry V, Tanner M, Blom T, Tynninen O,
Roselli A, Ollikainen M, Sariola H, Wartiovaara K and Nupponen NN:
Copy number alterations of the polycomb gene BMI1 in gliomas. Acta
Neuropathol. 116:97–102. 2008. View Article : Google Scholar : PubMed/NCBI
|
14
|
Louis DN, Ohgaki H, Wiestler OD, Cavenee
WK, Burger PC, Jouvet A, Scheithauer BW and Kleihues P: The 2007
WHO classification of tumours of the central nervous system. Acta
Neuropathol. 114:97–109. 2007. View Article : Google Scholar : PubMed/NCBI
|
15
|
Hemmati HD, Nakano I, Lazareff JA,
Masterman-Smith M, Geschwind DH, Bronner-Fraser M and Kornblum HI:
Cancerous stem cells can arise from pediatric brain tumors. Proc
Natl Acad Sci USA. 100:15178–15183. 2003. View Article : Google Scholar : PubMed/NCBI
|
16
|
Singh SK, Hawkins C, Clarke ID, Squire JA,
Bayani J, Hide T, Henkelman RM, Cusimano MD and Dirks PB:
Identification of human brain tumour initiating cells. Nature.
432:396–401. 2004. View Article : Google Scholar : PubMed/NCBI
|
17
|
Tso CL, Shintaku P, Chen J, Liu Q, Liu J,
Chen Z, Yoshimoto K, Mischel PS, Cloughesy TF, Liau LM and Nelson
SF: Primary glioblastomas express mesenchymal stem-like properties.
Mol Cancer Res. 4:607–619. 2006. View Article : Google Scholar : PubMed/NCBI
|
18
|
Chen R, Nishimura MC, Bumbaca SM,
Kharbanda S, Forrest WF, Kasman IM, Greve JM, Soriano RH, Gilmour
LL, Rivers CS, et al: A hierarchy of self-renewing tumor-initiating
cell types in glioblastoma. Cancer Cell. 17:362–375. 2010.
View Article : Google Scholar : PubMed/NCBI
|
19
|
Huber GF, Albinger-Hegyi A, Soltermann A,
Roessle M, Graf N, Haerle SK, Holzmann D, Moch H and Hegyi I:
Expression patterns of Bmi-1 and p16 significantly correlate with
overall, disease-specific and recurrence-free survival in
oropharyngeal squamous cell carcinoma. Cancer. 117:4659–4670. 2011.
View Article : Google Scholar : PubMed/NCBI
|
20
|
Vaissière T, Sawan C and Herceg Z:
Epigenetic interplay between histone modifications and DNA
methylation in gene silencing. Mutat Res. 659:40–48. 2008.
View Article : Google Scholar : PubMed/NCBI
|
21
|
Park IK, Morrison SJ and Clarke MF: Bmi1,
stem cells and senescence regulation. J Clin Invest. 113:175–179.
2004. View Article : Google Scholar : PubMed/NCBI
|
22
|
Wang E, Bhattacharyya S, Szabolcs A,
Rodriguez-Aguayo C, Jennings NB, Lopez-Berestein G, Mukherjee P,
Sood AK and Bhattacharya R: Enhancing chemotherapy response with
Bmi-1 silencing in ovarian cancer. PLoS One. 6:e179182011.
View Article : Google Scholar : PubMed/NCBI
|
23
|
Liu S, Dontu G, Mantle ID, Patel S, Ahn
NS, Jackson KW, Suri P and Wicha MS: Hedgehog signaling and Bmi-1
regulate self-renewal of normal and malignant human mammary stem
cells. Cancer Res. 66:6063–6071. 2006. View Article : Google Scholar : PubMed/NCBI
|
24
|
Song LB, Li J, Liao WT, Feng Y, Yu CP, Hu
LJ, Kong QL, Xu LH, Zhang X, Liu WL, et al: The polycomb group
protein Bmi-1 represses the tumor suppressor PTEN and induces
epithelial-mesenchymal transition in human nasopharyngeal
epithelial cells. J Clin Invest. 119:3626–3636. 2009. View Article : Google Scholar : PubMed/NCBI
|
25
|
Li J, Gong LY, Song LB, Jiang LL, Liu LP,
Wu J, Yuan J, Cai JC, He M, Wang L, et al: Oncoprotein Bmi-1
renders apoptotic resistance to glioma cells through activation of
the IKK-nuclear factor-κB pathway. Am J Pathol. 176:699–709. 2010.
View Article : Google Scholar : PubMed/NCBI
|
26
|
Beà S, Tort F, Pinyol M, Puig X, Hernández
L, Hernández S, Fernandez PL, van Lohuizen M, Colomer D and Campo
E: BMI-1 gene amplification and overexpression in hematological
malignancies occur mainly in mantle cell lymphomas. Cancer Res.
61:2409–2412. 2001.PubMed/NCBI
|
27
|
Cenci T, Martini M, Montano N,
D'Alessandris QG, Falchetti ML, Annibali D, Savino M, Bianchi F,
Pierconti F, Nasi S, et al: Prognostic relevance of c-Myc and BMI1
expression in patients with glioblastoma. Am J Clin Pathol.
138:390–396. 2012. View Article : Google Scholar : PubMed/NCBI
|
28
|
Farivar S, Keikha R Zati, Shiari R and
Jadali F: Expression of bmi-1 in pediatric brain tumors as a new
independent prognostic marker of patient survival. Biomed Res Int.
2013:1925482013. View Article : Google Scholar : PubMed/NCBI
|
29
|
Wu Z, Wang Q, Wang L, Li G, Liu H, Fan F,
Li Z, Li Y and Tu Y: Combined aberrant expression of Bmi1 and EZH2
is predictive of poor prognosis in glioma patients. J Neurol Sci.
335:191–196. 2013. View Article : Google Scholar : PubMed/NCBI
|
30
|
Molofsky AV, He S, Bydon M, Morrison SJ
and Pardal R: Bmi-1 promotes neural stem cell self-renewal and
neural development but not mouse growth and survival by repressing
the p16Ink4a and p19Arf senescence pathways. Genes Dev.
19:1432–1437. 2005. View Article : Google Scholar : PubMed/NCBI
|
31
|
Bommi PV, Dimri M, Sahasrabuddhe AA,
Khandekar J and Dimri GP: The polycomb group protein BMI1 is a
transcriptional target of HDAC inhibitors. Cell Cycle. 9:2663–2673.
2010. View Article : Google Scholar : PubMed/NCBI
|