1
|
Du CY, Shi YQ, Zhou Y, Fu H and Zhao G:
The analysis of status and clinical implication of KIT and PDGFRA
mutations in gastrointestinal stromal tumor (GIST). J Surg Oncol.
98:175–178. 2008. View Article : Google Scholar : PubMed/NCBI
|
2
|
Mavroeidis L, Metaxa-Mariatou V,
Papoudou-Bai A, Lampraki AM, Kostadima L, Tsinokou I, Zarkavelis G,
Papadaki A, Petrakis D, Gκoura S, et al: Comprehensive molecular
screening by next generation sequencing reveals a distinctive
mutational profile of KIT/PDGFRA genes and novel genomic
alterations: Results from a 20-year cohort of patients with GIST
from north-western Greece. ESMO Open. 3:e0003352018. View Article : Google Scholar : PubMed/NCBI
|
3
|
Lopes LF and Bacchi CE: Imatinib treatment
for gastrointestinal stromal tumour (GIST). J Cell Mol Med.
14:42–50. 2010. View Article : Google Scholar : PubMed/NCBI
|
4
|
Sanchez Hidalgo JM, Rufian Peña S, Ciria
Bru R, Naranjo Torres A, Muñoz Casares C, Ruiz Rabelo J and Briceño
Delgado J: Gastrointestinal stromal tumors (GIST): A prospective
evaluation of risk factors and prognostic scores. J Gastrointest
Cancer. 41:27–37. 2010. View Article : Google Scholar : PubMed/NCBI
|
5
|
Liu X, Qiu H, Wu Z, Zhang P, Feng X, Chen
T, Li Y, Tao K, Li G, Sun X, et al: A Novel pathological prognostic
score (PPS) to identify ‘very high-risk’ patients: A multicenter
retrospective analysis of 506 patients with high risk
gastrointestinal stromal tumor (GIST). J Gastrointest Surg.
22:2150–2157. 2018. View Article : Google Scholar : PubMed/NCBI
|
6
|
Liu Q, Li Y, Dong M, Kong F and Dong Q:
Gastrointestinal bleeding is an independent risk factor for poor
prognosis in GIST patients. Biomed Res Int.
2017:71524062017.PubMed/NCBI
|
7
|
Stegmüller J, Konishi Y, Huynh MA, Yuan Z,
Dibacco S and Bonni A: Cell-intrinsic regulation of axonal
morphogenesis by the Cdh1-APC target SnoN. Neuron. 50:389–400.
2006. View Article : Google Scholar : PubMed/NCBI
|
8
|
Pearson-White S, Deacon D, Crittenden R,
Brady G, Iscove N and Quesenberry PJ: The ski/sno protooncogene
family in hematopoietic development. Blood. 86:2146–2155. 1995.
View Article : Google Scholar : PubMed/NCBI
|
9
|
Sutrave P, Kelly AM and Hughes SH: Ski can
cause selective growth of skeletal muscle in transgenic mice. Genes
Dev. 4:1462–1472. 1990. View Article : Google Scholar : PubMed/NCBI
|
10
|
Liu X, Zhang E, Li P, Liu J, Zhou P, Gu
DY, Chen X, Cheng T and Zhou Y: Expression and possible mechanism
of c-ski, a novel tissue repair-related gene during normal and
radiation-impaired wound healing. Wound Repair Regen. 14:162–171.
2006. View Article : Google Scholar : PubMed/NCBI
|
11
|
Bravou V, Antonacopoulou A, Papadaki H,
Floratou K, Stavropoulos M, Episkopou V, Petropoulou C and
Kalofonos H: TGF-beta repressors SnoN and Ski are implicated in
human colorectal carcinogenesis. Cell Oncol. 31:41–51.
2009.PubMed/NCBI
|
12
|
Kiyono K, Suzuki HI, Morishita Y, Komuro
A, Iwata C, Yashiro M, Hirakawa K, Kano MR and Miyazono K: c-Ski
overexpression promotes tumor growth and angiogenesis through
inhibition of transforming growth factor-beta signaling in
diffuse-type gastric carcinoma. Cancer Sci. 100:1809–1816. 2009.
View Article : Google Scholar : PubMed/NCBI
|
13
|
Song L, Chen X, Gao S, Zhang C, Qu C, Wang
P and Liu L: Ski modulate the characteristics of pancreatic cancer
stem cells via regulating sonic hedgehog signaling pathway. Tumour
Biol. Oct 12–2016.(Epub ahead of print). View Article : Google Scholar
|
14
|
Yang H, Zhan L, Yang T, Wang L, Li C, Zhao
J, Lei Z, Li X and Zhang HT: Ski prevents TGF-β-induced EMT and
cell invasion by repressing SMAD-dependent signaling in non-small
cell lung cancer. Oncol Rep. 34:87–94. 2015. View Article : Google Scholar : PubMed/NCBI
|
15
|
Fletcher CD, Berman JJ, Corless C,
Gorstein F, Lasota J, Longley BJ, Miettinen M, O'Leary TJ, Remotti
H, Rubin BP, et al: Diagnosis of gastrointestinal stromal tumors: A
consensus approach. Hum Pathol. 33:459–465. 2002. View Article : Google Scholar : PubMed/NCBI
|
16
|
Patel S: Navigating risk stratification
systems for the management of patients with GIST. Ann Surg Oncol.
18:1698–1704. 2011. View Article : Google Scholar : PubMed/NCBI
|
17
|
Wozniak A, Rutkowski P, Piskorz A,
Ciwoniuk M, Osuch C, Bylina E, Sygut J, Chosia M, Rys J, Urbanczyk
K, et al: Prognostic value of KIT/PDGFRA mutations in
gastrointestinal stromal tumours (GIST): Polish Clinical GIST
Registry experience. Ann Oncol. 23:353–360. 2012. View Article : Google Scholar : PubMed/NCBI
|
18
|
Wardelmann E, Losen I, Hans V, Neidt I,
Speidel N, Bierhoff E, Heinicke T, Pietsch T, Büttner R and
Merkelbach-Bruse S: Deletion of Trp-557 and Lys-558 in the
juxtamembrane domain of the c-kit protooncogene is associated with
metastatic behavior of gastrointestinal stromal tumors. Int J
Cancer. 106:887–895. 2003. View Article : Google Scholar : PubMed/NCBI
|
19
|
Martín J, Poveda A, Llombart-Bosch A,
Ramos R, López-Guerrero JA, García del Muro J, Maurel J, Calabuig
S, Gutierrez A, González de Sande JL, et al: Deletions affecting
codons 557–558 of the c-KIT gene indicate a poor prognosis in
patients with completely resected gastrointestinal stromal tumors:
A study by the Spanish Group for Sarcoma Research (GEIS). J Clin
Oncol. 23:6190–6198. 2005. View Article : Google Scholar : PubMed/NCBI
|
20
|
Nishida T, Cho H, Hirota S, Masuzawa T,
Chiguchi G and Tsujinaka T; Kinki GIST Study Group, :
Clinicopathological features and prognosis of primary GISTs with
tumor rupture in the real world. Ann Surg Oncol. 25:1961–1969.
2018. View Article : Google Scholar : PubMed/NCBI
|
21
|
Asare EA and Feig BW: Raining Frogs,
flying horses, and defining tumor rupture in GIST. Ann Surg Oncol.
26:1601–1603. 2019. View Article : Google Scholar : PubMed/NCBI
|
22
|
Fukuchi M, Nakajima M, Fukai Y, Miyazaki
T, Masuda N, Sohda M, Manda R, Tsukada K, Kato H and Kuwano H:
Increased expression of c-Ski as a co-repressor in transforming
growth factor-beta signaling correlates with progression of
esophageal squamous cell carcinoma. Int J Cancer. 108:818–824.
2004. View Article : Google Scholar : PubMed/NCBI
|
23
|
Villanacci V, Bellone G, Battaglia E,
Rossi E, Carbone A, Prati A, Verna C, Niola P, Morelli A, Grassini
M and Bassotti G: Ski/SnoN expression in the sequence
metaplasia-dysplasia-adenocarcinoma of Barrett's esophagus. Hum
Pathol. 39:403–409. 2008. View Article : Google Scholar : PubMed/NCBI
|
24
|
Ritter M, Kattmann D, Teichler S, Hartmann
O, Samuelsson MK, Burchert A, Bach JP, Kim TD, Berwanger B, Thiede
C, et al: Inhibition of retinoic acid receptor signaling by Ski in
acute myeloid leukemia. Leukemia. 20:437–443. 2006. View Article : Google Scholar : PubMed/NCBI
|
25
|
Heider TR, Lyman S, Schoonhoven R and
Behrns KE: Ski promotes tumor growth through abrogation of
transforming growth factor-beta signaling in pancreatic cancer. Ann
Surg. 246:61–68. 2007. View Article : Google Scholar : PubMed/NCBI
|
26
|
Wang P, Chen Z, Meng ZQ, Fan J, Luo JM,
Liang W, Lin JH, Zhou ZH, Chen H, Wang K, et al: Dual role of Ski
in pancreatic cancer cells: Tumor-promoting versus
metastasis-suppressive function. Carcinogenesis. 30:1497–1506.
2009. View Article : Google Scholar : PubMed/NCBI
|
27
|
Boone B, Haspeslagh M and Brochez L:
Clinical significance of the expression of c-Ski and SnoN, possible
mediators in TGF-beta resistance, in primary cutaneous melanoma. J
Dermatol Sci. 53:26–33. 2009. View Article : Google Scholar : PubMed/NCBI
|